INT213993

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Context Info
Confidence 0.02
First Reported 2007
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 0.37
Pain Relevance 0.07

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (Spnb1) protein complex (Spnb1) cytoplasm (Spnb1)
Spnb1 (Mus musculus)
Pain Link Frequency Relevance Heat
sodium channel 2 75.04 Quite High
aspirin 3 5.00 Very Low Very Low Very Low
Somatostatin 3 5.00 Very Low Very Low Very Low
Inflammation 2 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Pain 1 5.00 Very Low Very Low Very Low
imagery 1 5.00 Very Low Very Low Very Low
antagonist 1 5.00 Very Low Very Low Very Low
Catecholamine 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Spherocytosis 1 99.42 Very High Very High Very High
Aging 21 83.64 Quite High
Syndrome 8 82.16 Quite High
Heart Arrhythmia 1 54.72 Quite High
Arrhythmia Under Development 22 50.24 Quite High
Death 8 50.00 Quite Low
Infection 8 13.16 Low Low
Myocardial Infarction 7 8.56 Low Low
Alopecia 3 5.00 Very Low Very Low Very Low
Bradycardia 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Collectively, these results indicate that downregulation of the JA biosynthesis pathway is not essential for SA-mediated suppression of JA signaling.
Negative_regulation (downregulation) of JA
1) Confidence 0.02 Published 2010 Journal Planta Section Body Doc Link PMC2957573 Disease Relevance 0 Pain Relevance 0
Consistent with this function, mutations in ankyrin-R are the major cause of hereditary spherocytosis, a disorder associated with spectrin deficiency and increased membrane fragility in humans [4]–[6].
Negative_regulation (deficiency) of spectrin associated with spherocytosis
2) Confidence 0.01 Published 2007 Journal PLoS ONE Section Body Doc Link PMC2013943 Disease Relevance 0.37 Pain Relevance 0.07

General Comments

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