INT215396

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Context Info
Confidence 0.03
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.23
Pain Relevance 0.23

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

lipid binding (Ppara) extracellular space (Il4) extracellular region (Il4)
nucleus (Ppara) DNA binding (Ppara) lipid metabolic process (Ppara)
Anatomy Link Frequency
peritoneal macrophages 2
Il4 (Mus musculus)
Ppara (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammation 34 100.00 Very High Very High Very High
cytokine 12 100.00 Very High Very High Very High
agonist 25 78.88 Quite High
COX-2 inhibitor 1 55.04 Quite High
Inflammatory response 5 5.00 Very Low Very Low Very Low
metalloproteinase 4 5.00 Very Low Very Low Very Low
fibrosis 3 5.00 Very Low Very Low Very Low
chemokine 2 5.00 Very Low Very Low Very Low
antagonist 2 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 37 100.00 Very High Very High Very High
Leukemia 1 9.24 Low Low
Asthma 8 5.00 Very Low Very Low Very Low
Pulmonary Disease 5 5.00 Very Low Very Low Very Low
Apoptosis 5 5.00 Very Low Very Low Very Low
Disease 5 5.00 Very Low Very Low Very Low
Pulmonary Alveolar Proteinosis 4 5.00 Very Low Very Low Very Low
Cancer 4 5.00 Very Low Very Low Very Low
Sarcoidosis 4 5.00 Very Low Very Low Very Low
Injury 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In peritoneal macrophages, the anti-inflammatory cytokine IL-4 upregulates expression of both 12/15-lipoxygenase and PPAR-?
anti-inflammatory cytokine IL-4 Positive_regulation (upregulates) of Gene_expression (expression) of PPAR in peritoneal macrophages associated with inflammation and cytokine
1) Confidence 0.03 Published 2007 Journal PPAR Research Section Body Doc Link PMC2066181 Disease Relevance 0.23 Pain Relevance 0.23

General Comments

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