INT216152

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Context Info
Confidence 0.23
First Reported 2007
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 0.41
Pain Relevance 0.15

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleolus (MTF1) nucleus (MTF1) intracellular (MTF1)
DNA binding (MTF1)
MTF1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Glutamate 1 97.28 Very High Very High Very High
metalloproteinase 1 94.44 High High
cytokine 18 59.44 Quite High
Inflammation 31 57.52 Quite High
psoriasis 22 5.00 Very Low Very Low Very Low
chemokine 9 5.00 Very Low Very Low Very Low
Inflammatory response 6 5.00 Very Low Very Low Very Low
Inflammatory stimuli 2 5.00 Very Low Very Low Very Low
methotrexate 1 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Wound Healing 1 88.16 High High
Targeted Disruption 1 84.32 Quite High
Apoptosis 7 62.92 Quite High
INFLAMMATION 37 57.52 Quite High
Necrosis 2 57.40 Quite High
Cancer 10 57.04 Quite High
Stress 34 49.20 Quite Low
Adhesions 2 45.76 Quite Low
Death 1 24.96 Low Low
Aging 1 24.52 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The MTF1 binding site enrichment was highest for the third gene set with five of the 11 genes containing the MRE element (Figure 3D).
MTF1 Binding (binding) of
1) Confidence 0.23 Published 2007 Journal PLoS Genetics Section Body Doc Link PMC2082467 Disease Relevance 0.23 Pain Relevance 0.06
The major ARE-binding transcription factor is nuclear factor-erythroid 2-related factor 2 (Nrf2), which, through heteromeric interaction with the small Maf proteins, binds the ARE and initiates the de novo expression of detoxifying enzymes [11].
ARE-binding transcription factor Binding (initiates) of
2) Confidence 0.09 Published 2010 Journal Mediators of Inflammation Section Body Doc Link PMC2929514 Disease Relevance 0.17 Pain Relevance 0.10

General Comments

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