INT217070

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Context Info
Confidence 0.25
First Reported 2007
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 8
Total Number 8
Disease Relevance 6.27
Pain Relevance 1.75

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Acsm3) molecular_function (Acsm3) cellular_component (Acsm3)
ligase activity (Acsm3) biological_process (Acsm3) lipid metabolic process (Acsm3)
Anatomy Link Frequency
brain 1
basilar artery 1
Acsm3 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
cva 371 100.00 Very High Very High Very High
cytokine 45 99.98 Very High Very High Very High
headache 3 98.60 Very High Very High Very High
anesthesia 24 96.96 Very High Very High Very High
ketamine 10 90.64 High High
withdrawal 5 82.36 Quite High
imagery 12 73.04 Quite High
Inflammation 60 70.24 Quite High
alcohol 52 67.08 Quite High
Inflammatory response 20 55.36 Quite High
Disease Link Frequency Relevance Heat
Subarachnoid Hemorrhage 315 100.00 Very High Very High Very High
Cv General 4 Under Development 11 100.00 Very High Very High Very High
Generalized Anxiety Disorder 24 99.98 Very High Very High Very High
Arterial-venous Cerebral Malformations 9 99.98 Very High Very High Very High
Convulsion 6 99.00 Very High Very High Very High
Hemorrhage 37 98.92 Very High Very High Very High
Headache 4 98.60 Very High Very High Very High
Affective Disorder 30 98.16 Very High Very High Very High
Cerebral Aneurysm 5 93.44 High High
Drug Dependence 72 89.36 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Patients with an AVM present with an SAH, neurological deficits, seizures, headaches or may be completely asymptomatic.
Gene_expression (present) of SAH associated with convulsion, headache, cva and arterial-venous cerebral malformations
1) Confidence 0.25 Published 2010 Journal Anesthesiology Research and Practice Section Body Doc Link PMC2911602 Disease Relevance 2.15 Pain Relevance 0.21
Experimental SAH was produced according to our previous study [11].
Gene_expression (produced) of SAH associated with cva
2) Confidence 0.25 Published 2009 Journal Mediators of Inflammation Section Body Doc Link PMC2821769 Disease Relevance 0.46 Pain Relevance 0.25
As revealed by Ersahin [7], treatment with melatonin due to its free radical scavenging properties significantly inhibited SAH-induced lipid peroxidation and neutrophil infiltration of the brain tissue on the second day of SAH induction, while the depleted antioxidant GSH level and inhibited Na+-K+-ATPase activity were restored to control levels.
Gene_expression (induction) of SAH in brain associated with cva
3) Confidence 0.25 Published 2009 Journal Mediators of Inflammation Section Body Doc Link PMC2821769 Disease Relevance 1.02 Pain Relevance 0.36
A total of 48 rabbits were randomly divided into four groups: control group, SAH group, SAH + vehicle group, and SAH + melatonin group.
Gene_expression (group) of SAH
4) Confidence 0.22 Published 2009 Journal Mediators of Inflammation Section Abstract Doc Link PMC2821769 Disease Relevance 0.40 Pain Relevance 0.18
B) activity, proinflammatory cytokines expression, and oxidative response in the basilar artery after SAH.
Gene_expression (expression) of SAH in basilar artery associated with cytokine
5) Confidence 0.22 Published 2009 Journal Mediators of Inflammation Section Abstract Doc Link PMC2821769 Disease Relevance 0.33 Pain Relevance 0.17
Forty-eight hours after the first SAH, a second one was produced in the same manner as the first.
Gene_expression (produced) of SAH associated with cva
6) Confidence 0.20 Published 2009 Journal Mediators of Inflammation Section Body Doc Link PMC2821769 Disease Relevance 0.61 Pain Relevance 0.48
Table 4 shows that, in the first block, lifetime dysthymia (OR = 2.8, p = 0.029) increased the risk of SA in the follow-up period, while lifetime GAD reduced the risk of SA in the same period (OR = 0.2, p = 0.045), when socio-demographic variables and number of PDs were controlled for.
Gene_expression (reduced) of SA associated with generalized anxiety disorder and affective disorder
7) Confidence 0.08 Published 2007 Journal Clin Pract Epidemiol Ment Health Section Body Doc Link PMC2098764 Disease Relevance 0.91 Pain Relevance 0
In the multivariate analyses, there was only a tendency towards an association between low education and SA.
Gene_expression (education) of SA
8) Confidence 0.08 Published 2007 Journal Clin Pract Epidemiol Ment Health Section Body Doc Link PMC2098764 Disease Relevance 0.38 Pain Relevance 0.10

General Comments

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