INT217305

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Context Info
Confidence 0.96
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.32
Pain Relevance 0.03

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular region (Acan) cell adhesion (Acan) proteinaceous extracellular matrix (Acan)
Acan (Mus musculus)
Pain Link Frequency Relevance Heat
5HT 1 59.00 Quite High
Dorsal column 27 47.28 Quite Low
Spinal cord 15 45.80 Quite Low
dorsal root ganglion 31 31.96 Quite Low
Sciatic nerve 50 18.80 Low Low
Peripheral nerve injury 5 5.00 Very Low Very Low Very Low
withdrawal 5 5.00 Very Low Very Low Very Low
halothane 3 5.00 Very Low Very Low Very Low
unmyelinated 3 5.00 Very Low Very Low Very Low
alcohol 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Injury 47 97.76 Very High Very High Very High
Targeted Disruption 79 61.88 Quite High
Cicatrix 4 57.80 Quite High
Ganglion Cysts 32 31.96 Quite Low
Nervous System Injury 15 5.00 Very Low Very Low Very Low
Peripheral Arterial Disease 7 5.00 Very Low Very Low Very Low
Overdose 6 5.00 Very Low Very Low Very Low
Sprains And Strains 2 5.00 Very Low Very Low Very Low
Frailty 2 5.00 Very Low Very Low Very Low
Facial Nerve Injuries 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
NG2 is the most strongly upregulated CSPG at CNS injury sites, with a time course very closely matched to the arrested growth of axonal sprouts [13,27], and enzymatic degradation of CSPGs (in particular, removal of chondroitin sulphate glycosaminoglycans by chondroitinase ABC) promotes regenerative axonal growth in the adult CNS [29-31].
Protein_catabolism (degradation) of CSPG associated with injury
1) Confidence 0.96 Published 2007 Journal BMC Neurosci Section Body Doc Link PMC2100060 Disease Relevance 0.32 Pain Relevance 0.03

General Comments

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