INT217758

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Context Info
Confidence 0.00
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 2
Disease Relevance 0.69
Pain Relevance 0.82

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Kcnd3) plasma membrane (Kcnd3) transmembrane transport (Kcnd3)
Kcnd3 (Mus musculus)
Pain Link Frequency Relevance Heat
diclofenac 56 93.60 High High
cINOD 38 90.40 High High
gABA 4 87.64 High High
Glutamate 4 87.28 High High
dorsal root ganglion 36 80.68 Quite High
Neuropathic pain 2 76.92 Quite High
Migraine 2 75.68 Quite High
hyperexcitability 8 75.12 Quite High
Neuronal excitability 4 48.60 Quite Low
potassium channel 2 40.80 Quite Low
Disease Link Frequency Relevance Heat
Convulsion 20 96.12 Very High Very High Very High
Ganglion Cysts 36 80.68 Quite High
Disease 6 80.68 Quite High
Neuropathic Pain 2 76.92 Quite High
Epilepsy 4 76.08 Quite High
Headache 2 75.68 Quite High
Benign Neonatal Epilepsy 2 66.88 Quite High
Myoclonus 2 65.12 Quite High
INFLAMMATION 8 47.16 Quite Low
Colon Cancer 14 8.96 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Conversion of the carboxylate moiety into amide successfully removes the COX inhibition but preserves the M-channel modulation.
Regulation (modulation) of M-channel
1) Confidence 0.00 Published 2007 Journal PLoS ONE Section Body Doc Link PMC2131780 Disease Relevance 0.51 Pain Relevance 0.47
Conversion of the carboxylate function into amide abolished COX inhibition but preserved M-channel modulation.
Regulation (modulation) of M-channel
2) Confidence 0.00 Published 2007 Journal PLoS ONE Section Abstract Doc Link PMC2131780 Disease Relevance 0.18 Pain Relevance 0.35

General Comments

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