INT218289

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.65
First Reported 2008
Last Reported 2008
Negated 1
Speculated 0
Reported most in Body
Documents 1
Total Number 3
Disease Relevance 1.44
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

kinase activity (Acvr2a) cytoplasm (Acvr2a)
Anatomy Link Frequency
muscle 3
Acvr2a (Mus musculus)
Pain Link Frequency Relevance Heat
anesthesia 3 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Targeted Disruption 15 100.00 Very High Very High Very High
Hypertrophy 6 98.96 Very High Very High Very High
Aging 3 5.00 Very Low Very Low Very Low
Confusion 3 5.00 Very Low Very Low Very Low
Muscular Atrophy 3 5.00 Very Low Very Low Very Low
Hyperinsulinism 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Hemizygous knockout of ACVR2B accentuates muscle growth in ACVR2A-knockout mice, although it has no effect in mice with normal ACVR2A expression [38].
Gene_expression (expression) of ACVR2A in muscle associated with targeted disruption
1) Confidence 0.65 Published 2008 Journal PLoS ONE Section Body Doc Link PMC2148100 Disease Relevance 0.44 Pain Relevance 0
The microarrays indicated that the ACVR2A gene is expressed in human muscle, with no significant difference between men and women.
Neg (no) Gene_expression (expressed) of ACVR2A in muscle
2) Confidence 0.65 Published 2008 Journal PLoS ONE Section Body Doc Link PMC2148100 Disease Relevance 0.43 Pain Relevance 0
In mice, knockout of either activin A receptor IIA (ACVR2A) or ACVR2B causes muscle hypertrophy [38].
Gene_expression (knockout) of ACVR2A in muscle associated with targeted disruption and hypertrophy
3) Confidence 0.56 Published 2008 Journal PLoS ONE Section Body Doc Link PMC2148100 Disease Relevance 0.57 Pain Relevance 0

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox