INT219793

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Context Info
Confidence 0.13
First Reported 2007
Last Reported 2011
Negated 0
Speculated 0
Reported most in Body
Documents 5
Total Number 5
Disease Relevance 3.42
Pain Relevance 0.61

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

isomerase activity (Renbp)
Anatomy Link Frequency
plaque 1
corpus callosum 1
Renbp (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Lasting pain 60 98.32 Very High Very High Very High
alcohol 18 87.92 High High
depression 4 73.60 Quite High
backache 1 27.44 Quite Low
withdrawal 1 22.00 Low Low
rheumatoid arthritis 1 18.56 Low Low
Chronic low back pain 1 6.56 Low Low
Pain 26 5.00 Very Low Very Low Very Low
drug abuse 7 5.00 Very Low Very Low Very Low
Inflammation 4 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Injury 46 100.00 Very High Very High Very High
Insulin Resistance 1 99.68 Very High Very High Very High
Obesity 2 98.82 Very High Very High Very High
Pain 86 98.32 Very High Very High Very High
Hyperlipidemia 1 97.32 Very High Very High Very High
Atherosclerosis 9 96.40 Very High Very High Very High
Hypertension 3 95.96 Very High Very High Very High
Adhesions 1 94.88 High High
Frailty 91 93.56 High High
Diabetes Mellitus 22 90.88 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
This result is not that surprising given the fact that chronic pain has a negative impact on physical function [27, 29, 49] and that physical function decreases with age [31].
Negative_regulation (decreases) of age associated with lasting pain
1) Confidence 0.13 Published 2010 Journal Qual Life Res Section Body Doc Link PMC2977061 Disease Relevance 0.84 Pain Relevance 0.61
The study found a significant decline of 1.4% annually for age-adjusted rates in women, but the rates remained stable in men.40 In neighbouring Austria, between 1994 and 2006, after adjustment for age and sex, the incidence rates rose from 471/100 000 to 567/100 000 per year in men and from 637/100 000 to 759/100 000 per year in women.41 This rise is predominantly accounted for by the rise seen in patients aged over 80 years.
Negative_regulation (decline) of age-adjusted
2) Confidence 0.11 Published 2011 Journal Indian Journal of Orthopaedics Section Body Doc Link PMC3004072 Disease Relevance 0.41 Pain Relevance 0
Studies in experimental obese Zucker rats, a model of insulin resistance induced by the AGE formation inhibitor pyridoxamine, have shown marked attenuation of the characteristic hyperlipidemia, reduced AGEs, and hypertension, indicating interactions between AGEs and lipids.22) One of the seminal in vivo studies performed in experimental diabetes-associated atherosclerosis demonstrated attenuation of plaque formation by the soluble receptor for AGEs (sRAGE).21)
Negative_regulation (inhibitor) of AGE in plaque associated with atherosclerosis, diabetes mellitus, hypertension, obesity, hyperlipidemia and insulin resistance
3) Confidence 0.08 Published 2010 Journal Korean Circulation Journal Section Body Doc Link PMC2978295 Disease Relevance 1.49 Pain Relevance 0
An Age at Treatment × Treatment interaction [F(2,57) = 3.2, P < .05], indicated that corpus callosum size decreased as the age of injury decreased from P5 to P1.
Negative_regulation (decreased) of age in corpus callosum associated with injury
4) Confidence 0.08 Published 2007 Journal BMC Neurosci Section Abstract Doc Link PMC2204005 Disease Relevance 0.46 Pain Relevance 0
The numbers of these sex-predominant genes at each age are presented in Figure 4.
Negative_regulation (presented) of age
5) Confidence 0.08 Published 2010 Journal BMC Genomics Section Body Doc Link PMC3012673 Disease Relevance 0.14 Pain Relevance 0

General Comments

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