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Context Info
Confidence 0.15
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.36
Pain Relevance 0.63

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

aging (EDNRA) cell-cell signaling (Edn1) cytoplasm (Edn1)
signal transducer activity (EDNRA) cell proliferation (EDNRA) signal transduction (EDNRA)
Anatomy Link Frequency
sensory neurons 1
EDNRA (Homo sapiens)
Edn1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
algogenic 4 100.00 Very High Very High Very High
antagonist 4 90.60 High High
agonist 5 89.96 High High
qutenza 40 85.68 High High
dorsal root ganglion 22 71.68 Quite High
adenocard 1 65.36 Quite High
Analgesic 2 65.12 Quite High
addiction 1 56.48 Quite High
tetrodotoxin 3 53.92 Quite High
nociceptor 6 42.60 Quite Low
Disease Link Frequency Relevance Heat
Nociception 7 88.36 High High
Ganglion Cysts 23 71.68 Quite High
Hyperalgesia 5 63.40 Quite High
Neuropathic Pain 4 10.64 Low Low
Pain 4 5.00 Very Low Very Low Very Low
INFLAMMATION 2 5.00 Very Low Very Low Very Low
Reprotox - General 1 1 5.00 Very Low Very Low Very Low
Cancer 1 5.00 Very Low Very Low Very Low
Inflammatory Pain 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Our results indicate that the ETAR is expressed in sensory neurons and could contribute to the algogenic effects of ET-1 by sensitizing TRPV1.
ETAR Negative_regulation (effects) of ET-1 in sensory neurons associated with algogenic
1) Confidence 0.15 Published 2007 Journal Mol Pain Section Body Doc Link PMC2206006 Disease Relevance 0.36 Pain Relevance 0.63

General Comments

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