INT219920

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.14
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.13

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (EDNRA, Trpv1) cytosol (Trpv1) cell proliferation (EDNRA)
signal transduction (EDNRA) aging (EDNRA) lipid metabolic process (Trpv1)
EDNRA (Homo sapiens)
Trpv1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
qutenza 40 99.98 Very High Very High Very High
ASIC 1 67.52 Quite High
dorsal root ganglion 22 5.00 Very Low Very Low Very Low
bradykinin 10 5.00 Very Low Very Low Very Low
nociceptor 6 5.00 Very Low Very Low Very Low
Pain 5 5.00 Very Low Very Low Very Low
agonist 5 5.00 Very Low Very Low Very Low
algogenic 4 5.00 Very Low Very Low Very Low
antagonist 4 5.00 Very Low Very Low Very Low
tetrodotoxin 3 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Ganglion Cysts 23 5.00 Very Low Very Low Very Low
Nociception 7 5.00 Very Low Very Low Very Low
Hyperalgesia 5 5.00 Very Low Very Low Very Low
Pain 4 5.00 Very Low Very Low Very Low
Neuropathic Pain 4 5.00 Very Low Very Low Very Low
INFLAMMATION 2 5.00 Very Low Very Low Very Low
Reprotox - General 1 1 5.00 Very Low Very Low Very Low
Cancer 1 5.00 Very Low Very Low Very Low
Inflammatory Pain 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
HEK293 cells transfected with the same amounts of TRPV1 and the ETBR showed a less prominent ET-1-induced potentiation of capsaicin-stimulated TRPV1 currents when compared to that elicited by the ETAR (Fig. 4).
ETAR Positive_regulation (stimulated) of TRPV1 associated with qutenza
1) Confidence 0.14 Published 2007 Journal Mol Pain Section Body Doc Link PMC2206006 Disease Relevance 0 Pain Relevance 0.13

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox