INT219921

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Context Info
Confidence 0.13
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.36
Pain Relevance 0.63

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (EDNRA, Trpv1) cytosol (Trpv1) cell proliferation (EDNRA)
signal transduction (EDNRA) aging (EDNRA) lipid metabolic process (Trpv1)
Anatomy Link Frequency
sensory neurons 1
EDNRA (Homo sapiens)
Trpv1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
algogenic 4 100.00 Very High Very High Very High
antagonist 4 90.60 High High
agonist 5 89.96 High High
qutenza 40 87.36 High High
dorsal root ganglion 22 73.36 Quite High
Analgesic 2 66.80 Quite High
adenocard 1 65.36 Quite High
addiction 1 56.48 Quite High
tetrodotoxin 3 53.92 Quite High
nociceptor 6 42.60 Quite Low
Disease Link Frequency Relevance Heat
Nociception 7 88.36 High High
Ganglion Cysts 23 73.36 Quite High
Hyperalgesia 5 63.40 Quite High
Neuropathic Pain 4 12.32 Low Low
Pain 4 5.00 Very Low Very Low Very Low
INFLAMMATION 2 5.00 Very Low Very Low Very Low
Reprotox - General 1 1 5.00 Very Low Very Low Very Low
Cancer 1 5.00 Very Low Very Low Very Low
Inflammatory Pain 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Our results indicate that the ETAR is expressed in sensory neurons and could contribute to the algogenic effects of ET-1 by sensitizing TRPV1.
ETAR Negative_regulation (sensitizing) of TRPV1 in sensory neurons associated with algogenic
1) Confidence 0.13 Published 2007 Journal Mol Pain Section Body Doc Link PMC2206006 Disease Relevance 0.36 Pain Relevance 0.63

General Comments

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