INT221217

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Context Info
Confidence 0.18
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.93
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Vdac1) mitochondrion (Vdac1) plasma membrane (Vdac1)
protein complex (Vdac1) transmembrane transport (Vdac1)
Anatomy Link Frequency
liver 1
Vdac1 (Mus musculus)
Pain Link Frequency Relevance Heat
potassium channel 76 5.00 Very Low Very Low Very Low
Kinase C 38 5.00 Very Low Very Low Very Low
adenocard 4 5.00 Very Low Very Low Very Low
action potential duration 4 5.00 Very Low Very Low Very Low
ischemia 3 5.00 Very Low Very Low Very Low
bradykinin 3 5.00 Very Low Very Low Very Low
agonist 2 5.00 Very Low Very Low Very Low
noradrenaline 1 5.00 Very Low Very Low Very Low
addiction 1 5.00 Very Low Very Low Very Low
Action potential 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Parkinson's Disease 116 99.36 Very High Very High Very High
Reperfusion Injury 30 85.28 High High
Cv Unclassified Under Development 68 80.40 Quite High
Targeted Disruption 8 51.56 Quite High
Stress 19 14.48 Low Low
Apoptosis 16 5.00 Very Low Very Low Very Low
Injury 6 5.00 Very Low Very Low Very Low
Death 5 5.00 Very Low Very Low Very Low
Thrombosis Related Under Development 4 5.00 Very Low Very Low Very Low
Pressure And Volume Under Development 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
However, the recent demonstration that liver mitochondria from mice lacking VDAC1 exhibit normal MPTP opening and inhibition by ubiquinone analogues [67] make an essential role for VDAC1 unlikely.
Negative_regulation (lacking) of VDAC1 in liver associated with parkinson's disease
1) Confidence 0.18 Published 2007 Journal Biochimica et Biophysica Acta Section Body Doc Link PMC2212780 Disease Relevance 0.93 Pain Relevance 0

General Comments

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