INT221240

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Context Info
Confidence 0.00
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.89
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (Akt1, Bcl2) cytoplasm (Akt1, Bcl2) cytosol (Akt1, Bcl2)
mitochondrion (Bcl2) transport (Akt1) aging (Akt1)
Akt1 (Mus musculus)
Bcl2 (Mus musculus)
Pain Link Frequency Relevance Heat
potassium channel 76 5.00 Very Low Very Low Very Low
Kinase C 38 5.00 Very Low Very Low Very Low
adenocard 4 5.00 Very Low Very Low Very Low
action potential duration 4 5.00 Very Low Very Low Very Low
ischemia 3 5.00 Very Low Very Low Very Low
bradykinin 3 5.00 Very Low Very Low Very Low
agonist 2 5.00 Very Low Very Low Very Low
noradrenaline 1 5.00 Very Low Very Low Very Low
addiction 1 5.00 Very Low Very Low Very Low
Action potential 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 16 100.00 Very High Very High Very High
Cv Unclassified Under Development 68 79.60 Quite High
Parkinson's Disease 116 68.84 Quite High
Death 5 58.64 Quite High
Rupture 2 50.64 Quite High
Pressure And Volume Under Development 4 50.16 Quite High
Reperfusion Injury 30 5.00 Very Low Very Low Very Low
Stress 19 5.00 Very Low Very Low Very Low
Targeted Disruption 8 5.00 Very Low Very Low Very Low
Injury 6 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
This is brought about by Akt-mediated phosphorylation of the pro-apoptotic Bcl-2 family member Bad [273,274] and, via GSK3 phosphorylation and stabilisation of the anti-apoptotic Bcl-2 family member Mcl-1 [275].


Akt Positive_regulation (mediated) of Phosphorylation (phosphorylation) of Bcl-2 family member associated with apoptosis
1) Confidence 0.00 Published 2007 Journal Biochimica et Biophysica Acta Section Body Doc Link PMC2212780 Disease Relevance 0.89 Pain Relevance 0

General Comments

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