INT221632

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Context Info
Confidence 0.02
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.58
Pain Relevance 0.15

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

lipid binding (Fes) signal transduction (Plxna1) Golgi apparatus (Fes)
plasma membrane (Fes) cytoskeleton (Fes) intracellular (Plxna1)
Plxna1 (Mus musculus)
Fes (Mus musculus)
Pain Link Frequency Relevance Heat
dorsal root ganglion 74 93.92 High High
withdrawal 2 5.00 Very Low Very Low Very Low
Root ganglion neuron 2 5.00 Very Low Very Low Very Low
nociceptor 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Ganglion Cysts 76 93.92 High High
Shock 44 89.92 High High
Sprains And Strains 4 34.60 Quite Low
Apoptosis 6 5.00 Very Low Very Low Very Low
Targeted Disruption 6 5.00 Very Low Very Low Very Low
Death 2 5.00 Very Low Very Low Very Low
Adhesions 2 5.00 Very Low Very Low Very Low
Wallerian Degeneration 2 5.00 Very Low Very Low Very Low
Urological Neuroanatomy 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
PlexinA1 inhibits the autophosphorylation activities of Fps and Fer
PlexinA1 Negative_regulation (inhibits) of Fps
1) Confidence 0.02 Published 2007 Journal BMC Dev Biol Section Body Doc Link PMC2217550 Disease Relevance 0.17 Pain Relevance 0.08
CRMP2 was a substrate for Fps and Fer; and both CRMP2 and PlexinA1 inhibited Fps and Fer activities in vitro.
PlexinA1 Negative_regulation (inhibited) of Fps
2) Confidence 0.02 Published 2007 Journal BMC Dev Biol Section Body Doc Link PMC2217550 Disease Relevance 0.41 Pain Relevance 0.07

General Comments

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