INT221635

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Context Info
Confidence 0.20
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (Dpysl2, Fes) cell differentiation (Dpysl2) hydrolase activity, acting on carbon-nitrogen (but not peptide) bonds (Dpysl2)
mitochondrion (Dpysl2) Golgi apparatus (Fes) protein complex (Dpysl2)
Dpysl2 (Mus musculus)
Fes (Mus musculus)
Pain Link Frequency Relevance Heat
dorsal root ganglion 37 33.96 Quite Low
nociceptor 1 25.60 Quite Low
withdrawal 1 5.00 Very Low Very Low Very Low
Root ganglion neuron 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Ganglion Cysts 38 50.00 Quite Low
Shock 22 5.00 Very Low Very Low Very Low
Apoptosis 3 5.00 Very Low Very Low Very Low
Targeted Disruption 3 5.00 Very Low Very Low Very Low
Sprains And Strains 2 5.00 Very Low Very Low Very Low
Death 1 5.00 Very Low Very Low Very Low
Adhesions 1 5.00 Very Low Very Low Very Low
Wallerian Degeneration 1 5.00 Very Low Very Low Very Low
Urological Neuroanatomy 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Given the apparent ability of PlexinA1 and CRMP2 to inhibit Fps and Fer autophosphorylation, we conclude that some interaction must have been occurring, but it failed to survive our immunoprecipitation protocol.
CRMP2 Negative_regulation (inhibit) of Phosphorylation (autophosphorylation) of Fps
1) Confidence 0.20 Published 2007 Journal BMC Dev Biol Section Body Doc Link PMC2217550 Disease Relevance 0 Pain Relevance 0

General Comments

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