INT222493

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Context Info
Confidence 0.10
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0
Pain Relevance 0.71

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular region (Edn1, Prl) extracellular space (Edn1) cell-cell signaling (Edn1)
cytoplasm (Edn1)
Edn1 (Rattus norvegicus)
Prl (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Dopamine 26 92.08 High High
antagonist 78 80.88 Quite High
dopamine receptor 4 78.20 Quite High
Nerve growth factor 56 75.08 Quite High
Inflammation 54 5.00 Very Low Very Low Very Low
cytokine 44 5.00 Very Low Very Low Very Low
agonist 44 5.00 Very Low Very Low Very Low
Calcitonin gene-related peptide 34 5.00 Very Low Very Low Very Low
substance P 32 5.00 Very Low Very Low Very Low
gABA 30 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Stress 96 47.68 Quite Low
INFLAMMATION 56 5.00 Very Low Very Low Very Low
Cancer 44 5.00 Very Low Very Low Very Low
Targeted Disruption 44 5.00 Very Low Very Low Very Low
Pituitary Cancer 22 5.00 Very Low Very Low Very Low
Starvation 20 5.00 Very Low Very Low Very Low
Rheumatoid Arthritis 16 5.00 Very Low Very Low Very Low
Natriuresis 14 5.00 Very Low Very Low Very Low
Adhesions 12 5.00 Very Low Very Low Very Low
Hyperplasia 12 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The prominent action of exogenous ET is profound inhibition of PRL secretion (268, 399–401) but, when studied in a perifused cell column, ET-1 initially induces a prompt and short-lasting increase in PRL release, which is followed by a profound sustained inhibition (402).
ET-1 Positive_regulation (induces) of Localization (release) of PRL
1) Confidence 0.10 Published 2008 Journal Journal of Neuroendocrinology Section Body Doc Link PMC2229370 Disease Relevance 0 Pain Relevance 0.35
The prominent action of exogenous ET is profound inhibition of PRL secretion (268, 399–401) but, when studied in a perifused cell column, ET-1 initially induces a prompt and short-lasting increase in PRL release, which is followed by a profound sustained inhibition (402).
ET-1 Positive_regulation (increase) of Localization (release) of PRL
2) Confidence 0.10 Published 2008 Journal Journal of Neuroendocrinology Section Body Doc Link PMC2229370 Disease Relevance 0 Pain Relevance 0.35

General Comments

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