INT223444

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Context Info
Confidence 0.28
First Reported 2007
Last Reported 2009
Negated 1
Speculated 0
Reported most in Body
Documents 3
Total Number 5
Disease Relevance 2.95
Pain Relevance 0.52

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Anatomy Link Frequency
CD86 1
dendritic cells 1
skin 1
T cells 1
Mat1a (Mus musculus)
Pain Link Frequency Relevance Heat
adenocard 2 96.84 Very High Very High Very High
Neuropeptide 3 92.24 High High
addiction 1 89.76 High High
Neurotransmitter 1 81.84 Quite High
nociceptor 18 74.00 Quite High
Central nervous system 1 72.12 Quite High
Pain 5 59.76 Quite High
Congenital analgesia 1 54.00 Quite High
substance P 1 37.84 Quite Low
cytokine 18 10.68 Low Low
Disease Link Frequency Relevance Heat
Hypersensitivity 5 100.00 Very High Very High Very High
Disease 110 99.40 Very High Very High Very High
Infection 129 83.48 Quite High
Respiratory Disease 9 82.60 Quite High
Neuroblastoma 3 81.68 Quite High
Severe Acute Respiratory Syndrome 114 75.00 Quite High
Increased Venous Pressure Under Development 2 66.20 Quite High
Hyperhomocysteinemia 5 58.08 Quite High
Cognitive Disorder 7 55.40 Quite High
Syndrome 1 54.80 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Indeed, there are studies showing a role for AdoMet and AdoHcy in AD [2, 24, 30].
AdoMet Binding (role) of associated with disease
1) Confidence 0.28 Published 2009 Journal Neurol Sci Section Body Doc Link PMC2746292 Disease Relevance 0.63 Pain Relevance 0.13
Inhibitory AMs are believed to prevent the development of immune responses to environmental antigens and allergic responses by interacting with lung dendritic cells and T cells.
AMs Binding (interacting) of in T cells associated with hypersensitivity
2) Confidence 0.20 Published 2009 Journal PLoS Pathogens Section Abstract Doc Link PMC2762542 Disease Relevance 0.93 Pain Relevance 0
When cultured in the absence of AMs, aDC upregulated expression of CD86, MHC II and CD40.
AMs Neg (absence) Binding (absence) of in CD86
3) Confidence 0.18 Published 2009 Journal PLoS Pathogens Section Body Doc Link PMC2762542 Disease Relevance 0.35 Pain Relevance 0
For example, slowly adapting mechanoreceptors [SAMs] that interact with Merkel cells in the skin and whose phenotypic development depends on BDNF/TrkB signaling [3] can only be identified using physiological criteria.
SAMs Binding (interact) of in skin
4) Confidence 0.17 Published 2007 Journal BMC Neurosci Section Body Doc Link PMC2241628 Disease Relevance 0.11 Pain Relevance 0.39
Inhibitory AMs are believed to prevent the development of immune responses to environmental antigens and allergic responses by interacting with lung dendritic cells and T cells.
AMs Binding (interacting) of in dendritic cells associated with hypersensitivity
5) Confidence 0.07 Published 2009 Journal PLoS Pathogens Section Abstract Doc Link PMC2762542 Disease Relevance 0.93 Pain Relevance 0

General Comments

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