INT223513

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Context Info
Confidence 0.03
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 5
Disease Relevance 3.52
Pain Relevance 0.21

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleolus (Imp3) RNA binding (Imp3) rRNA binding (Imp3)
nucleus (Imp3) intracellular (Imp3) ribosome biogenesis (Imp3)
Imp3 (Mus musculus)
Pain Link Frequency Relevance Heat
Multiple sclerosis 15 95.36 Very High Very High Very High
anesthesia 40 85.60 High High
Demyelination 5 74.96 Quite High
rheumatoid arthritis 10 5.00 Very Low Very Low Very Low
Inflammation 10 5.00 Very Low Very Low Very Low
Inflammatory response 5 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Burns 455 99.98 Very High Very High Very High
Sepsis 10 95.60 Very High Very High Very High
Demyelinating Disease 20 95.36 Very High Very High Very High
Stress 65 94.84 High High
Disease 35 90.84 High High
Systemic Lupus Erythematosus 10 86.16 High High
Murine Acquired Immunodeficiency Syndrome 10 85.44 High High
Hematological Disease 5 85.16 High High
Diabetes Mellitus 5 84.80 Quite High
Pathologic Processes 5 83.64 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
It might be necessary to further characterize the biological roles of MuERVs presumed to be derived from U3 probes induced after burn.
Positive_regulation (induced) of U3 associated with burns
1) Confidence 0.03 Published 2007 Journal BMC Genomics Section Body Doc Link PMC2241634 Disease Relevance 0.33 Pain Relevance 0.05
The putative MuERVs cloned by U3 probes, which were presumed to originate from the burn-induced U3 fragments, will be the primary focus for future studies.


Positive_regulation (induced) of U3 associated with burns
2) Confidence 0.03 Published 2007 Journal BMC Genomics Section Body Doc Link PMC2241634 Disease Relevance 0.10 Pain Relevance 0
These MuERVs had unique U3 promoter sequences suggesting different profiles of transcription regulatory elements in each of these sequences.
Positive_regulation (sequences) of U3
3) Confidence 0.03 Published 2007 Journal BMC Genomics Section Body Doc Link PMC2241634 Disease Relevance 1.49 Pain Relevance 0.08
The L-1-1 U3 and U-1-8 U3 in the group II were likely to be derived from the baseline expression of the ~450 bp fragment expressed in no burn mice (3 hours).
Positive_regulation (derived) of U3 associated with burns
4) Confidence 0.03 Published 2007 Journal BMC Genomics Section Body Doc Link PMC2241634 Disease Relevance 0.68 Pain Relevance 0.09
Phylogenetic analysis revealed that four of the U3 sequences (K-4-5 U3, K-4-10 U3, L-4-3 U3, and U-4-1 U3) whose transcriptional activities are presumed to be induced in burn mice were clustered into a unique branch (Figure 3) and all have the HMGA1/2 binding element.
Positive_regulation (induced) of U3 associated with burns
5) Confidence 0.03 Published 2007 Journal BMC Genomics Section Body Doc Link PMC2241634 Disease Relevance 0.91 Pain Relevance 0

General Comments

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