INT223867

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Context Info
Confidence 0.25
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.87

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (Camk2a) kinase activity (Camk2a) cytoplasm (Camk2a)
Anatomy Link Frequency
neurons 2
Camk2a (Mus musculus)
Ltp (Mus musculus)
Pain Link Frequency Relevance Heat
long-term potentiation 104 100.00 Very High Very High Very High
Hippocampus 9 88.04 High High
Anterior cingulate cortex 34 82.84 Quite High
Kinase C 1 79.52 Quite High
Lasting pain 4 29.36 Quite Low
allodynia 1 14.40 Low Low
nMDA receptor 14 5.00 Very Low Very Low Very Low
Action potential 6 5.00 Very Low Very Low Very Low
Pyramidal cell 3 5.00 Very Low Very Low Very Low
amygdala 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Pain 5 29.36 Quite Low
Targeted Disruption 1 23.84 Low Low
Neuropathic Pain 1 14.40 Low Low
Anxiety Disorder 6 9.84 Low Low
Injury 1 5.00 Very Low Very Low Very Low
Stress 1 5.00 Very Low Very Low Very Low
Disseminated Intravascular Coagulation 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Calcium influx into the postsynaptic membrane is an essential process in the induction of LTP [54], and this enhanced calcium levels in the postsynaptic neurons activate several protein kinases including the CaMKII, which plays a pivotal role in the induction of LTP [38,55,56].
CaMKII Positive_regulation (role) of Positive_regulation (induction) of LTP in neurons associated with long-term potentiation
1) Confidence 0.25 Published 2007 Journal Mol Pain Section Body Doc Link PMC2245920 Disease Relevance 0 Pain Relevance 0.87

General Comments

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