INT224826

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Context Info
Confidence 0.55
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 11
Disease Relevance 1.94
Pain Relevance 2.57

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (SALL1) intracellular (SALL1) DNA binding (SALL1)
cytoplasm (SALL1)
Anatomy Link Frequency
cortex 1
synapse 1
SALL1 (Homo sapiens)
Pain Link Frequency Relevance Heat
nMDA receptor 11 99.16 Very High Very High Very High
Lasting pain 44 89.16 High High
Pain threshold 66 87.32 High High
Pain 451 85.84 High High
primary somatosensory cortex 517 76.96 Quite High
Acute pain 55 70.96 Quite High
qutenza 11 66.28 Quite High
c fibre 11 64.04 Quite High
Transcranial magnetic stimulation 77 48.80 Quite Low
Thalamus 11 46.92 Quite Low
Disease Link Frequency Relevance Heat
Pain 583 89.16 High High
Nociception 33 45.08 Quite Low
Epilepsy 11 17.60 Low Low
Cognitive Disorder 22 5.00 Very Low Very Low Very Low
Syndrome 11 5.00 Very Low Very Low Very Low
Fatigue 11 5.00 Very Low Very Low Very Low
Mental Disorders 11 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Recently Huang et al. (2007) have demonstrated that the after-effects produced by both iTBS or cTBS are NMDA-receptor dependent and hence they are more likely to involve plasticity-like changes at the synapse in the M1.
Gene_expression (produced) of iTBS in synapse
1) Confidence 0.55 Published 2007 Journal Exp Brain Res Section Body Doc Link PMC2248215 Disease Relevance 0.24 Pain Relevance 0.28
The solid line shows LEPs before and the intermittent line after TBS interventions
Gene_expression (interventions) of TBS
2) Confidence 0.55 Published 2007 Journal Exp Brain Res Section Body Doc Link PMC2248215 Disease Relevance 0 Pain Relevance 0
The TBS was followed by a post-stimulation LEP recording in ?
Gene_expression (followed) of TBS
3) Confidence 0.55 Published 2007 Journal Exp Brain Res Section Body Doc Link PMC2248215 Disease Relevance 0.07 Pain Relevance 0.07
15–20 min) and the TBS were applied for 40–190 s through the cap.
Gene_expression (applied) of TBS
4) Confidence 0.55 Published 2007 Journal Exp Brain Res Section Body Doc Link PMC2248215 Disease Relevance 0.07 Pain Relevance 0.07
It is possible that the different effectiveness observed between TBS protocols on motor and sensory cortices could be due to differences in the physiological and functional states of the stimulated cortex.
Gene_expression (protocols) of TBS in cortex
5) Confidence 0.55 Published 2007 Journal Exp Brain Res Section Body Doc Link PMC2248215 Disease Relevance 0.13 Pain Relevance 0.21
After TBS the impedance of the EEG electrodes were retested and corrected below 5 k?
Gene_expression (retested) of TBS
6) Confidence 0.55 Published 2007 Journal Exp Brain Res Section Body Doc Link PMC2248215 Disease Relevance 0.07 Pain Relevance 0.07
More recently, it was found that the excitatory effects of iTBS were reversed after NMDA receptor activation by D-cycloserin (Teo et al. 2007).
Gene_expression (effects) of iTBS associated with nmda receptor
7) Confidence 0.55 Published 2007 Journal Exp Brain Res Section Body Doc Link PMC2248215 Disease Relevance 0.16 Pain Relevance 0.21
In contrast, intermittent TBS (iTBS) facilitated M1 activity and produced increase in MEP amplitudes.
Gene_expression (produced) of iTBS
8) Confidence 0.55 Published 2007 Journal Exp Brain Res Section Body Doc Link PMC2248215 Disease Relevance 0.27 Pain Relevance 0.35
In contrast, intermittent TBS (iTBS) facilitated M1 activity and produced increase in MEP amplitudes.
Gene_expression (produced) of intermittent TBS
9) Confidence 0.55 Published 2007 Journal Exp Brain Res Section Body Doc Link PMC2248215 Disease Relevance 0.27 Pain Relevance 0.35
The results of the experiments with single trains of TBS suggest that in the human M1 TBS produces a mixture of facilitatory and inhibitory effects on synaptic transmission (Huang and Rothwell 2004).
Gene_expression (produces) of TBS
10) Confidence 0.55 Published 2007 Journal Exp Brain Res Section Body Doc Link PMC2248215 Disease Relevance 0.60 Pain Relevance 0.76
They found that cTBS increased phosphene thresholds whilst iTBS was found to be ineffective.
Gene_expression (ineffective) of iTBS
11) Confidence 0.55 Published 2007 Journal Exp Brain Res Section Body Doc Link PMC2248215 Disease Relevance 0.07 Pain Relevance 0.19

General Comments

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