INT224827

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Context Info
Confidence 0.37
First Reported 2007
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 5
Disease Relevance 3.36
Pain Relevance 0.62

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (SALL1) intracellular (SALL1) DNA binding (SALL1)
cytoplasm (SALL1)
Anatomy Link Frequency
monocytes 2
fibroblasts 2
SALL1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Somatosensory cortex 9 97.08 Very High Very High Very High
Acute pain 5 96.80 Very High Very High Very High
Lasting pain 4 95.64 Very High Very High Very High
primary somatosensory cortex 47 91.40 High High
Transcranial magnetic stimulation 7 83.40 Quite High
Pain 41 78.16 Quite High
cytokine 24 56.24 Quite High
nMDA receptor 1 43.96 Quite Low
imagery 28 5.00 Very Low Very Low Very Low
Pain threshold 6 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Muscular Dystrophy 8 98.70 Very High Very High Very High
Adhesions 12 97.08 Very High Very High Very High
Pain 53 96.80 Very High Very High Very High
Choroideremia 664 95.46 Very High Very High Very High
Stress 20 40.60 Quite Low
Disease 40 5.00 Very Low Very Low Very Low
Age-related Macular Degeneration 16 5.00 Very Low Very Low Very Low
Targeted Disruption 16 5.00 Very Low Very Low Very Low
Toxicity 12 5.00 Very Low Very Low Very Low
Necrosis 8 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Future studies should also clarify the effectiveness of the different TBS paradigms applied over the M1 and non-motor cortical areas, such as the SII on acute pain perception and in chronic pain.



Regulation (effectiveness) of TBS associated with somatosensory cortex, lasting pain and acute pain
1) Confidence 0.37 Published 2007 Journal Exp Brain Res Section Body Doc Link PMC2248215 Disease Relevance 0.34 Pain Relevance 0.51
Functional categories affected in both fibroblasts and monocytes of CHM patients included a large group of genes involved in regulation of cell adhesion and motility (NCAM-2, FMN1, ANKRD28, CSF3R, DMD, MLLT4, DPP4, TSPAN5, CD114, DMD, CYFIP2, WNT5A, MMP7), immune response (IL1, CC2, CFI, ELOVL2), regulation of trafficking and exocytosis (RGS11, SYT 6, GBF1) and transcriptional regulation (HOXD11, MRO, NRG1, NLRC3, NR2F6, SALL1, DIP2A, RSOPO2) (Fig. 7A).
Regulation (regulation) of SALL1 in monocytes associated with muscular dystrophy, choroideremia and adhesions
2) Confidence 0.06 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2793004 Disease Relevance 0.72 Pain Relevance 0.03
Functional categories affected in both fibroblasts and monocytes of CHM patients included a large group of genes involved in regulation of cell adhesion and motility (NCAM-2, FMN1, ANKRD28, CSF3R, DMD, MLLT4, DPP4, TSPAN5, CD114, DMD, CYFIP2, WNT5A, MMP7), immune response (IL1, CC2, CFI, ELOVL2), regulation of trafficking and exocytosis (RGS11, SYT 6, GBF1) and transcriptional regulation (HOXD11, MRO, NRG1, NLRC3, NR2F6, SALL1, DIP2A, RSOPO2) (Fig. 7A).
Regulation (regulation) of SALL1 in monocytes associated with muscular dystrophy, choroideremia and adhesions
3) Confidence 0.06 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2793004 Disease Relevance 0.79 Pain Relevance 0.03
Functional categories affected in both fibroblasts and monocytes of CHM patients included a large group of genes involved in regulation of cell adhesion and motility (NCAM-2, FMN1, ANKRD28, CSF3R, DMD, MLLT4, DPP4, TSPAN5, CD114, DMD, CYFIP2, WNT5A, MMP7), immune response (IL1, CC2, CFI, ELOVL2), regulation of trafficking and exocytosis (RGS11, SYT 6, GBF1) and transcriptional regulation (HOXD11, MRO, NRG1, NLRC3, NR2F6, SALL1, DIP2A, RSOPO2) (Fig. 7A).
Regulation (regulation) of SALL1 in fibroblasts associated with muscular dystrophy, choroideremia and adhesions
4) Confidence 0.02 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2793004 Disease Relevance 0.79 Pain Relevance 0.03
Functional categories affected in both fibroblasts and monocytes of CHM patients included a large group of genes involved in regulation of cell adhesion and motility (NCAM-2, FMN1, ANKRD28, CSF3R, DMD, MLLT4, DPP4, TSPAN5, CD114, DMD, CYFIP2, WNT5A, MMP7), immune response (IL1, CC2, CFI, ELOVL2), regulation of trafficking and exocytosis (RGS11, SYT 6, GBF1) and transcriptional regulation (HOXD11, MRO, NRG1, NLRC3, NR2F6, SALL1, DIP2A, RSOPO2) (Fig. 7A).
Regulation (regulation) of SALL1 in fibroblasts associated with muscular dystrophy, choroideremia and adhesions
5) Confidence 0.02 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2793004 Disease Relevance 0.72 Pain Relevance 0.03

General Comments

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