INT224829

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Context Info
Confidence 0.03
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.53
Pain Relevance 1.08

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (PKN2) cell adhesion (PKN2) cytoskeleton (PKN2)
nucleus (PKN2) intracellular (PKN2) cell cycle (PKN2)
Anatomy Link Frequency
ACC 1
PKN2 (Homo sapiens)
Pain Link Frequency Relevance Heat
Somatosensory cortex 18 99.46 Very High Very High Very High
primary somatosensory cortex 94 99.10 Very High Very High Very High
Anterior cingulate cortex 12 97.84 Very High Very High Very High
Pain 82 91.64 High High
Pain threshold 12 22.24 Low Low
nociceptor 2 21.12 Low Low
imagery 16 5.00 Very Low Very Low Very Low
Transcranial magnetic stimulation 14 5.00 Very Low Very Low Very Low
Acute pain 10 5.00 Very Low Very Low Very Low
Lasting pain 8 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Pain 106 91.64 High High
Cognitive Disorder 4 87.28 High High
Nociception 6 5.00 Very Low Very Low Very Low
Epilepsy 2 5.00 Very Low Very Low Very Low
Syndrome 2 5.00 Very Low Very Low Very Low
Fatigue 2 5.00 Very Low Very Low Very Low
Mental Disorders 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
However, the involvement of the contralateral SI and bilateral SII—parasylvian region in the N2 component generation was also reported (Kakigi et al. 2005; Kanda et al. 2000; Ohara et al. 2004).
Gene_expression (generation) of N2 associated with somatosensory cortex and primary somatosensory cortex
1) Confidence 0.03 Published 2007 Journal Exp Brain Res Section Body Doc Link PMC2248215 Disease Relevance 0.36 Pain Relevance 0.64
The N2 component (peaking around 160–220 ms), is generated bilaterally in the operculoinsular region and in the anterior cingulate cortex (ACC) (Garcia-Larrea et al. 2003) and reflects sensory, discriminatory processes (Garcia-Larrea et al. 1997; Iannetti et al. 2005); whilst the P2 component (peaking around 300–360 ms) arose mostly from the ACC and represents attentional, cognitive and affective factors of pain perception and processing (Treede 2003).
Gene_expression (generated) of N2 in ACC associated with pain, cognitive disorder and anterior cingulate cortex
2) Confidence 0.03 Published 2007 Journal Exp Brain Res Section Body Doc Link PMC2248215 Disease Relevance 0.17 Pain Relevance 0.44

General Comments

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