INT224833

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Context Info
Confidence 0.02
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.56
Pain Relevance 0.70

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (PKN2) cell adhesion (PKN2) cytoskeleton (PKN2)
nucleus (PKN2) intracellular (PKN2) cell cycle (PKN2)
PKN2 (Homo sapiens)
Pain Link Frequency Relevance Heat
Pain 41 99.38 Very High Very High Very High
primary somatosensory cortex 47 98.52 Very High Very High Very High
Pain threshold 6 91.68 High High
Somatosensory cortex 9 50.24 Quite High
Anterior cingulate cortex 6 39.12 Quite Low
insula 2 5.28 Low Low
imagery 8 5.00 Very Low Very Low Very Low
Transcranial magnetic stimulation 7 5.00 Very Low Very Low Very Low
Acute pain 5 5.00 Very Low Very Low Very Low
Lasting pain 4 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Pain 53 99.38 Very High Very High Very High
Nociception 3 8.56 Low Low
Cognitive Disorder 2 5.00 Very Low Very Low Very Low
Epilepsy 1 5.00 Very Low Very Low Very Low
Syndrome 1 5.00 Very Low Very Low Very Low
Fatigue 1 5.00 Very Low Very Low Very Low
Mental Disorders 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
One possibility is that modulation the excitability of the left SI by TBS activated some elements of the pain related network that caused a decrease in the N2 amplitude.
Negative_regulation (decrease) of N2 associated with pain and primary somatosensory cortex
1) Confidence 0.02 Published 2007 Journal Exp Brain Res Section Body Doc Link PMC2248215 Disease Relevance 0.56 Pain Relevance 0.70

General Comments

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