INT226209

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Context Info
Confidence 0.10
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 1.16
Pain Relevance 0.17

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (Pik3cb, Akt1) cytosol (Pik3cb, Akt1) signal transduction (Pik3cb, Akt1)
nucleus (Pik3cb, Akt1) transport (Akt1) aging (Akt1)
Pik3cb (Rattus norvegicus)
Akt1 (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammation 20 62.00 Quite High
cytokine 6 55.12 Quite High
Inflammatory mediators 4 5.00 Very Low Very Low Very Low
fibrosis 2 5.00 Very Low Very Low Very Low
Inflammatory marker 2 5.00 Very Low Very Low Very Low
Pain 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Frailty 38 91.44 High High
Hypertrophy 4 90.96 High High
Insulin Resistance 18 73.64 Quite High
INFLAMMATION 26 62.00 Quite High
Cancer 10 59.36 Quite High
Necrosis 2 58.88 Quite High
Chronic Renal Failure 56 50.00 Quite Low
Diabetes Mellitus 10 40.84 Quite Low
Acidosis 12 39.96 Quite Low
Sepsis 10 39.56 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In protein synthesis, insulin-like growth factor (IGF)-1 and insulin signaling leads to increased phosphatidylinositol 3-kinase (PI3K), which promotes Akt phosphorylation (Akt-P).
PI3K Positive_regulation (promotes) of Phosphorylation (phosphorylation) of Akt-P
1) Confidence 0.10 Published 2007 Journal Pediatr Nephrol Section Body Doc Link PMC2259254 Disease Relevance 0.58 Pain Relevance 0.09
In protein synthesis, insulin-like growth factor (IGF)-1 and insulin signaling leads to increased phosphatidylinositol 3-kinase (PI3K), which promotes Akt phosphorylation (Akt-P).
PI3K Positive_regulation (promotes) of Phosphorylation (phosphorylation) of Akt
2) Confidence 0.07 Published 2007 Journal Pediatr Nephrol Section Body Doc Link PMC2259254 Disease Relevance 0.58 Pain Relevance 0.09

General Comments

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