INT226344

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Context Info
Confidence 0.07
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.03
Pain Relevance 0.18

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (IL9, CCL5) extracellular region (IL9, CCL5) aging (CCL5)
cell-cell signaling (CCL5) cytoplasm (CCL5)
IL9 (Homo sapiens)
CCL5 (Homo sapiens)
Pain Link Frequency Relevance Heat
cytokine 91 97.36 Very High Very High Very High
Inflammation 96 79.92 Quite High
antagonist 34 48.52 Quite Low
chemokine 23 5.00 Very Low Very Low Very Low
agonist 20 5.00 Very Low Very Low Very Low
Inflammatory response 6 5.00 Very Low Very Low Very Low
methotrexate 4 5.00 Very Low Very Low Very Low
Inflammatory mediators 2 5.00 Very Low Very Low Very Low
withdrawal 2 5.00 Very Low Very Low Very Low
cINOD 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Eosinophilia 19 100.00 Very High Very High Very High
Asthma 315 99.90 Very High Very High Very High
Occupational Lung Diseases 15 97.28 Very High Very High Very High
Pneumonia 7 80.72 Quite High
Hyperplasia 9 73.76 Quite High
INFLAMMATION 103 62.68 Quite High
Targeted Disruption 20 5.00 Very Low Very Low Very Low
Disease 17 5.00 Very Low Very Low Very Low
Rhinitis 11 5.00 Very Low Very Low Very Low
Hypersensitivity 9 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In animal models of asthma the overexpression of IL-9 causes BAL eosinophilia, peribronchial accumulation of collagen and increased BAL levels of CCL5 and CTGF [220].
Gene_expression (overexpression) of IL-9 Positive_regulation (increased) of CCL5 associated with asthma and eosinophilia
1) Confidence 0.07 Published 2008 Journal J Occup Med Toxicol Section Body Doc Link PMC2259400 Disease Relevance 1.03 Pain Relevance 0.18

General Comments

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