INT226368

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.11
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.92
Pain Relevance 0.20

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (TNF) extracellular space (TNF) extracellular region (TNF)
plasma membrane (TNF) intracellular (TNF) extracellular matrix organization (TNF)
TNF (Homo sapiens)
IGHE (Homo sapiens)
Pain Link Frequency Relevance Heat
cytokine 91 100.00 Very High Very High Very High
Inflammation 96 90.16 High High
chemokine 23 80.64 Quite High
antagonist 34 53.84 Quite High
agonist 20 5.00 Very Low Very Low Very Low
Inflammatory response 6 5.00 Very Low Very Low Very Low
methotrexate 4 5.00 Very Low Very Low Very Low
Inflammatory mediators 2 5.00 Very Low Very Low Very Low
withdrawal 2 5.00 Very Low Very Low Very Low
cINOD 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Eosinophilia 19 100.00 Very High Very High Very High
Asthma 315 97.00 Very High Very High Very High
INFLAMMATION 103 90.16 High High
Occupational Lung Diseases 15 67.16 Quite High
Hypersensitivity 9 42.68 Quite Low
Pulmonary Disease 2 24.32 Low Low
Mycobacterial Infection 1 23.92 Low Low
Sarcoidosis 1 23.36 Low Low
Disease 17 22.08 Low Low
Apoptosis 9 15.20 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
On the other hand, in other animal models, administration of IL-18 enhances antigen-induced increase in serum IgE and Th2 cytokines and airway eosinophilia in part by increasing antigen-induced TNF?
Positive_regulation (increasing) of TNF Positive_regulation (enhances) of Positive_regulation (increase) of IgE associated with eosinophilia and cytokine
1) Confidence 0.11 Published 2008 Journal J Occup Med Toxicol Section Body Doc Link PMC2259400 Disease Relevance 0.92 Pain Relevance 0.20

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox