INT226507

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Context Info
Confidence 0.13
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.20
Pain Relevance 0.15

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Jun) signal transduction (Il1rl1) extracellular space (Il1rl1)
extracellular region (Il1rl1) nucleus (Jun) plasma membrane (Il1rl1)
Il1rl1 (Mus musculus)
Jun (Mus musculus)
Pain Link Frequency Relevance Heat
tolerance 1 99.12 Very High Very High Very High
cytokine 91 71.12 Quite High
Inflammation 96 25.08 Quite Low
antagonist 34 5.00 Very Low Very Low Very Low
chemokine 23 5.00 Very Low Very Low Very Low
agonist 20 5.00 Very Low Very Low Very Low
Inflammatory response 6 5.00 Very Low Very Low Very Low
methotrexate 4 5.00 Very Low Very Low Very Low
Inflammatory mediators 2 5.00 Very Low Very Low Very Low
withdrawal 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Eosinophilia 19 91.16 High High
Asthma 315 51.16 Quite High
Targeted Disruption 20 41.64 Quite Low
INFLAMMATION 103 25.08 Quite Low
Eczema 4 13.60 Low Low
Disease 17 5.00 Very Low Very Low Very Low
Occupational Lung Diseases 15 5.00 Very Low Very Low Very Low
Rhinitis 11 5.00 Very Low Very Low Very Low
Hypersensitivity 9 5.00 Very Low Very Low Very Low
Hyperplasia 9 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Additionally, ST2L is able to activate the transcription factor AP-1; increase phosphorylation of c-Jun, and activate the MAP kinases c-Jun N-terminal kinase (JNK), p42/p44 and p38.
ST2L Positive_regulation (activate) of Gene_expression (kinases) of AP
1) Confidence 0.13 Published 2008 Journal J Occup Med Toxicol Section Body Doc Link PMC2259400 Disease Relevance 0.20 Pain Relevance 0.15

General Comments

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