INT22721

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Context Info
Confidence 0.23
First Reported 1984
Last Reported 1990
Negated 1
Speculated 0
Reported most in Abstract
Documents 2
Total Number 2
Disease Relevance 0.17
Pain Relevance 0.53

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Acot1) mitochondrion (Acot1) lipid metabolic process (Acot1)
cytoplasm (Acot1)
Anatomy Link Frequency
motoneurons 2
Acot1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Dopamine 2 98.60 Very High Very High Very High
cocaine 1 89.40 High High
Desipramine 1 88.96 High High
Norepinephrine uptake inhibitor 1 88.48 High High
Clonidine 2 84.88 Quite High
Neurotransmitter 2 75.00 Quite High
tetrodotoxin 1 71.68 Quite High
cerebral cortex 1 68.40 Quite High
Disease Link Frequency Relevance Heat
Muscular Atrophy 1 96.48 Very High Very High Very High
Paralysis 1 70.00 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The results indicate that motoneurons control the stability of junctional AChRs through evoked muscle activity and the number of junctional AChRs through trophic factors.
Regulation (control) of Protein_catabolism (stability) of AChRs in motoneurons
1) Confidence 0.23 Published 1990 Journal Neuron Section Abstract Doc Link 2322461 Disease Relevance 0.17 Pain Relevance 0.07
The action of ACh was not modified by the presence of nomifensine, a dopamine uptake inhibitor.
Neg (not) Regulation (modified) of Protein_catabolism (action) of ACh associated with dopamine
2) Confidence 0.23 Published 1984 Journal J. Pharmacol. Exp. Ther. Section Abstract Doc Link 6150107 Disease Relevance 0 Pain Relevance 0.46

General Comments

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