INT227796

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Context Info
Confidence 0.16
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.16
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Acsl1, Nos3) plasma membrane (Acsl1, Nos3) Golgi apparatus (Nos3)
endoplasmic reticulum (Acsl1) peroxisome (Acsl1) cytoplasm (Nos3)
Acsl1 (Mus musculus)
Nos3 (Mus musculus)
Pain Link Frequency Relevance Heat
fibrosis 1 43.16 Quite Low
cva 7 5.00 Very Low Very Low Very Low
Angina 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Acute Coronary Syndrome 44 100.00 Very High Very High Very High
Myocardial Infarction 12 97.32 Very High Very High Very High
Coronary Artery Disease 56 92.04 High High
Hypertrophy 3 58.48 Quite High
Ventricular Remodeling 2 47.92 Quite Low
Cardiovascular Disorder Under Development 1 43.16 Quite Low
Cv General 3 Under Development 7 5.00 Very Low Very Low Very Low
Nicotine Addiction 7 5.00 Very Low Very Low Very Low
Hypertension 5 5.00 Very Low Very Low Very Low
Disorder Of Lipid Metabolism 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
These findings imply that, although the mechanism underlying the association between the eNOS gene polymorphism and ACS has so far remained elusive, the genetic background controlling nitric oxide may be associated with the pathogenesis of ACS.



ACS Binding (association) of eNOS associated with acute coronary syndrome
1) Confidence 0.16 Published 2008 Journal Lipids Health Dis Section Body Doc Link PMC2267783 Disease Relevance 1.16 Pain Relevance 0

General Comments

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