INT227935

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Context Info
Confidence 0.08
First Reported 2007
Last Reported 2009
Negated 1
Speculated 0
Reported most in Body
Documents 4
Total Number 7
Disease Relevance 0.51
Pain Relevance 0.94

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

pigmentation (Mlph) protein targeting (Mlph)
Anatomy Link Frequency
vessels 1
AsPC-1 1
amygdala 1
T lymphocytes 1
Mlph (Mus musculus)
Pain Link Frequency Relevance Heat
Pyramidal cell 85 98.60 Very High Very High Very High
amygdala 2 97.60 Very High Very High Very High
chemokine 1 91.88 High High
GABAergic 9 87.44 High High
Anterior cingulate cortex 52 85.12 High High
interneuron 53 76.20 Quite High
Inflammation 12 70.12 Quite High
depression 9 23.64 Low Low
Action potential 6 5.00 Very Low Very Low Very Low
Thalamus 5 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 148 98.12 Very High Very High Very High
Adhesions 41 75.00 Quite High
INFLAMMATION 6 70.12 Quite High
Pancreatic Cancer 192 50.00 Quite Low
Cytomegalovirus Infection 4 25.32 Quite Low
Depression 9 23.64 Low Low
Uveitis 3 22.32 Low Low
Eye Disease 2 18.80 Low Low
Cancer 68 5.00 Very Low Very Low Very Low
Metastasis 16 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In conclusion, our data demonstrate for the first time that the integrin-FAK signaling axis is activated in collateral vessels and that altered expression of FN and LN may play a crucial role in mediating the integrin-FAK signaling pathway activation.
Gene_expression (expression) of LN in vessels
1) Confidence 0.08 Published 2008 Journal Mol Cell Biochem Section Abstract Doc Link PMC2758386 Disease Relevance 0.14 Pain Relevance 0
Our results demonstrated that LN protected AsPC-1 cells from Gem-induced cytotoxicity in a time-dependent manner, and the protective effect was most obvious at 72 h after Gem treatment (Fig. 8A).
Gene_expression (protected) of LN in AsPC-1
2) Confidence 0.04 Published 2009 Journal Mol Cancer Section Body Doc Link PMC2806309 Disease Relevance 0.30 Pain Relevance 0
Cells were then distributed onto LN-coated (LN) or control (plastic) wells and cultured in SITA medium (RPMI 1640 supplemented with 30 nM selenium, 5 ?
Gene_expression (distributed) of LN
3) Confidence 0.04 Published 2009 Journal Mol Cancer Section Body Doc Link PMC2806309 Disease Relevance 0 Pain Relevance 0
Cells were then distributed onto LN-coated (LN) or control (plastic) wells and cultured in SITA medium (RPMI 1640 supplemented with 30 nM selenium, 5 ?
Gene_expression (distributed) of LN
4) Confidence 0.04 Published 2009 Journal Mol Cancer Section Body Doc Link PMC2806309 Disease Relevance 0 Pain Relevance 0
Cell culture plastics were coated with LN (10 ?
Gene_expression (coated) of LN
5) Confidence 0.04 Published 2009 Journal Mol Cancer Section Body Doc Link PMC2806309 Disease Relevance 0 Pain Relevance 0
In the cervical LN of VIP-Rh-Lip injected rats, VIP was detected in T lymphocytes.
Gene_expression (detected) of LN in T lymphocytes
6) Confidence 0.02 Published 2007 Journal Molecular Vision Section Body Doc Link PMC2268712 Disease Relevance 0.07 Pain Relevance 0.08
For example, a GluR2-lacking AMPA receptor is expressed mainly in interneurons but not in pyramidal neurons in the amygdala [32].
Neg (not) Gene_expression (expressed) of GluR2-lacking in amygdala associated with pyramidal cell and amygdala
7) Confidence 0.02 Published 2009 Journal Mol Brain Section Body Doc Link PMC2770551 Disease Relevance 0 Pain Relevance 0.86

General Comments

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