INT228456

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Context Info
Confidence 0.24
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.97
Pain Relevance 0.12

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Trpv1) transport (Trpv1) extracellular space (Igf1)
extracellular region (Igf1) plasma membrane (Trpv1) transmembrane transport (Trpv1)
Igf1 (Mus musculus)
Trpv1 (Mus musculus)
Pain Link Frequency Relevance Heat
Hyperalgesia 14 61.28 Quite High
dorsal root ganglion 42 56.12 Quite High
Pain 22 50.00 Quite Low
Spinal cord 6 35.24 Quite Low
Deafferentation 1 21.44 Low Low
diabetic neuropathy 17 5.00 Very Low Very Low Very Low
hypoalgesia 14 5.00 Very Low Very Low Very Low
qutenza 10 5.00 Very Low Very Low Very Low
Dopamine 3 5.00 Very Low Very Low Very Low
Thermal hyperalgesia 3 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Diabetes Mellitus 118 98.36 Very High Very High Very High
Hyperglycemia 8 90.36 High High
Hyperalgesia 48 78.88 Quite High
Targeted Disruption 36 57.36 Quite High
Ganglion Cysts 42 56.12 Quite High
Pain 21 50.00 Quite Low
Neuropathic Pain 8 47.28 Quite Low
Stress 1 15.60 Low Low
Peripheral Neuropathy 17 5.00 Very Low Very Low Very Low
Hypoalagesia 14 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Previously, we showed that insulin and IGF-1, acting via the IGF-1 receptor, potentiates TRPV1 current by activating PKC [50].
IGF-1 Positive_regulation (potentiates) of TRPV1
1) Confidence 0.24 Published 2008 Journal Mol Pain Section Body Doc Link PMC2275252 Disease Relevance 0.97 Pain Relevance 0.12

General Comments

This test has worked.

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