INT228976

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Context Info
Confidence 0.06
First Reported 2008
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 3
Total Number 4
Disease Relevance 3.45
Pain Relevance 0.17

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (MCAT)
Anatomy Link Frequency
fat 2
striatum 1
MCAT (Homo sapiens)
Pain Link Frequency Relevance Heat
withdrawal 4 84.68 Quite High
Inflammation 6 75.32 Quite High
corticosteroid 4 71.48 Quite High
Somatostatin 4 55.12 Quite High
adenocard 2 53.84 Quite High
medulla 2 29.32 Quite Low
Substantia nigra 2 28.52 Quite Low
cva 8 5.00 Very Low Very Low Very Low
Dopamine 4 5.00 Very Low Very Low Very Low
fibrosis 4 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Pressure And Volume Under Development 58 98.96 Very High Very High Very High
Disease 250 97.76 Very High Very High Very High
Waldmann Disease 138 97.76 Very High Very High Very High
Lymphedema 32 94.72 High High
Staphylococcus Infection 2 84.88 Quite High
Enteropathy 16 80.04 Quite High
Systemic Lupus Erythematosus 4 78.40 Quite High
INFLAMMATION 6 75.32 Quite High
Pleural Effusion 8 67.40 Quite High
Malignant Neoplastic Disease 8 61.04 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
They can be used after or in combination with a low-fat diet associated with MCT supplementation.
MCT Binding (associated) of in fat
1) Confidence 0.06 Published 2008 Journal Orphanet J Rare Dis Section Body Doc Link PMC2288596 Disease Relevance 0.64 Pain Relevance 0.04
PIL is a chronic debilitating disorder requiring constraining long-term dietary control based on a low-fat regimen associated with supplementary MCT.
MCT Binding (associated) of in fat associated with waldmann disease
2) Confidence 0.05 Published 2008 Journal Orphanet J Rare Dis Section Body Doc Link PMC2288596 Disease Relevance 1.50 Pain Relevance 0.10
Oxygen utilization and assembly of mtETC complexes were also determined.


mtETC Binding (complexes) of
3) Confidence 0.01 Published 2009 Journal Mol Neurodegener Section Abstract Doc Link PMC2711937 Disease Relevance 0.81 Pain Relevance 0.03
Movement, fission, and fusion of mitochondria are functions that are also essential for neuronal survival. [8] Studies at the ultrastructural level in human PD caudate and striatum detected a disruption in the distribution of both microtubules and organelles such as mitochondria. [9] Neurotoxins that are used to model PD pathogenesis (1-methyl-4-phenylpyridinium (MPP+) and rotenone) compromise mitochondrial function by inhibiting complex I of the mtETC.
mtETC Binding (complex) of in striatum associated with disease
4) Confidence 0.01 Published 2009 Journal Mol Neurodegener Section Body Doc Link PMC2711937 Disease Relevance 0.49 Pain Relevance 0

General Comments

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