INT229087

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Context Info
Confidence 0.03
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.86
Pain Relevance 0.33

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular region (IL1R1) biosynthetic process (ACCS) plasma membrane (IL1R1)
intracellular (IL1R1) signal transducer activity (IL1R1)
IL1R1 (Homo sapiens)
ACCS (Homo sapiens)
Pain Link Frequency Relevance Heat
antagonist 14 97.72 Very High Very High Very High
Inflammatory marker 3 95.44 Very High Very High Very High
Inflammation 35 90.76 High High
anakinra 4 70.48 Quite High
cytokine 12 5.00 Very Low Very Low Very Low
rheumatoid arthritis 6 5.00 Very Low Very Low Very Low
withdrawal 5 5.00 Very Low Very Low Very Low
depression 2 5.00 Very Low Very Low Very Low
Inflammatory response 2 5.00 Very Low Very Low Very Low
Arthritis 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Atherosclerosis 12 96.28 Very High Very High Very High
INFLAMMATION 38 95.12 Very High Very High Very High
Death 5 87.40 High High
Acute Coronary Syndrome 42 82.28 Quite High
Myocardial Infarction 7 77.20 Quite High
Adverse Drug Reaction 12 5.00 Very Low Very Low Very Low
Coronary Artery Disease 7 5.00 Very Low Very Low Very Low
Rheumatoid Arthritis 7 5.00 Very Low Very Low Very Low
Injury 5 5.00 Very Low Very Low Very Low
Pressure And Volume Under Development 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Pre-clinical studies indicate that IL-1 may be a primary driver of ACS and that the naturally occurring interleukin-1 receptor antagonist (IL-1ra) may inhibit the atherosclerotic process.
IL-1 Negative_regulation (driver) of ACS associated with atherosclerosis and antagonist
1) Confidence 0.03 Published 2008 Journal Trials Section Abstract Doc Link PMC2289802 Disease Relevance 0.86 Pain Relevance 0.33

General Comments

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