INT230715

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Context Info
Confidence 0.14
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.64
Pain Relevance 1.02

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

small molecule metabolic process (KCNB1) plasma membrane (KCNB1) transmembrane transport (KCNB1)
KCNB1 (Homo sapiens)
Pain Link Frequency Relevance Heat
potassium channel 5 99.82 Very High Very High Very High
Pain 14 94.04 High High
Ziconotide 4 89.32 High High
intrathecal 1 86.76 High High
Intractable pain 1 86.16 High High
Calcium channel 2 82.20 Quite High
Lasting pain 2 71.36 Quite High
sodium channel 21 70.48 Quite High
tricyclic antidepressant 4 68.36 Quite High
Nav1.7 24 59.40 Quite High
Disease Link Frequency Relevance Heat
Diabetes Mellitus 1 96.40 Very High Very High Very High
Pain 15 94.04 High High
Intractable Pain 1 86.16 High High
Hyperalgesia 1 66.84 Quite High
Diabetic Neuropathy 1 58.08 Quite High
Neuralgia 1 57.16 Quite High
Herpes Simplex Virus Infection 1 56.76 Quite High
Neuropathic Pain 13 52.44 Quite High
Targeted Disruption 4 5.00 Very Low Very Low Very Low
Nociception 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
cell delayed rectifying potassium channel, Kv2.1 (KCNB1) is a potential target for enhancing glucose-dependent insulin secretion, and thus for the treatment of type II diabetes (Herrington et al., 2006).
Regulation (target) of KCNB1 associated with diabetes mellitus and potassium channel
1) Confidence 0.14 Published 2008 Journal The Journal of General Physiology Section Body Doc Link PMC2346569 Disease Relevance 0.64 Pain Relevance 1.02

General Comments

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