INT230745

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Context Info
Confidence 0.05
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.22
Pain Relevance 0.29

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (Ace, Icam1) plasma membrane (Ace, Icam1) endosome (Ace)
peptidase activity (Ace) extracellular region (Ace) cell adhesion (Icam1)
Ace (Mus musculus)
Icam1 (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammation 41 92.24 High High
Inflammatory mediators 4 87.44 High High
Calcium channel 1 51.88 Quite High
Angina 2 5.00 Very Low Very Low Very Low
Inflammatory marker 2 5.00 Very Low Very Low Very Low
Bioavailability 2 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
metalloproteinase 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Diabetes Mellitus 7 99.14 Very High Very High Very High
Adhesions 6 96.20 Very High Very High Very High
Renal Disease 1 94.92 High High
INFLAMMATION 38 92.24 High High
Hypertension 32 84.64 Quite High
Cardiovascular Disease 11 46.64 Quite Low
Hyperlipidemia 4 39.28 Quite Low
Increased Venous Pressure Under Development 4 29.36 Quite Low
Insulin Resistance 2 15.32 Low Low
Weight Loss 4 6.24 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In type 2 diabetic patients, ARBs diminished VCAM-1, whereas ACE inhibitors reduced ICAM-1 (Guba et al 2000).
Negative_regulation (inhibitors) of ACE Negative_regulation (reduced) of ICAM-1 associated with diabetes mellitus
1) Confidence 0.05 Published 2007 Journal Vascular Health and Risk Management Section Body Doc Link PMC2350124 Disease Relevance 1.22 Pain Relevance 0.29

General Comments

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