INT230746

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Context Info
Confidence 0.16
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.27
Pain Relevance 0.09

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (Crp, Serpine1) extracellular region (Serpine1)
Anatomy Link Frequency
vasculature 2
Crp (Mus musculus)
Serpine1 (Mus musculus)
Pain Link Frequency Relevance Heat
Bioavailability 2 94.08 High High
Inflammation 41 82.00 Quite High
Angina 2 17.56 Low Low
Inflammatory mediators 4 5.00 Very Low Very Low Very Low
Inflammatory marker 2 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
metalloproteinase 1 5.00 Very Low Very Low Very Low
Calcium channel 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Adhesions 6 96.16 Very High Very High Very High
Atherosclerotic Plaque 1 87.80 High High
INFLAMMATION 38 82.00 Quite High
Hypertension 32 47.04 Quite Low
Stress 4 33.44 Quite Low
Vasculitis 10 31.72 Quite Low
Rupture 4 21.08 Low Low
Cv General 3 Under Development 2 17.56 Low Low
Cardiovascular Disease 11 5.00 Very Low Very Low Very Low
Diabetes Mellitus 7 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In endothelial cells, CRP facilitated the release of PAI-1 (Devaraj et al 2003) and endothelin-1 (Verma et al 2002), and increased the expression of cell adhesion molecules (Pasceri et al 2000), reduced NO bioavailability (Venugopal et al 2002), and NO-mediated dilation in the vasculature.
CRP Positive_regulation (facilitated) of Localization (release) of PAI-1 in vasculature associated with adhesions and bioavailability
1) Confidence 0.16 Published 2007 Journal Vascular Health and Risk Management Section Body Doc Link PMC2350124 Disease Relevance 0.27 Pain Relevance 0.09

General Comments

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