INT230974

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Context Info
Confidence 0.49
First Reported 2008
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 0
Pain Relevance 0.57

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (GRIA3) plasma membrane (GRIA3) protein complex (GRIA3)
Anatomy Link Frequency
neuron 2
GRIA3 (Homo sapiens)
Pain Link Frequency Relevance Heat
Glutamate receptor 12 100.00 Very High Very High Very High
Dopamine 2 93.68 High High
Neurotransmitter 9 90.16 High High
agonist 3 90.08 High High
Glutamate 19 88.64 High High
antagonist 9 87.52 High High
Action potential 2 80.40 Quite High
substance P 1 75.40 Quite High
Migraine 46 50.00 Quite Low
depression 6 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Headache 45 50.00 Quite Low
Neurodegenerative Disease 9 39.24 Quite Low
Disease 7 6.56 Low Low
Depression 6 5.00 Very Low Very Low Very Low
Epilepsy 6 5.00 Very Low Very Low Very Low
Obesity 3 5.00 Very Low Very Low Very Low
Migraine With Aura 3 5.00 Very Low Very Low Very Low
Neuritis 2 5.00 Very Low Very Low Very Low
Death 2 5.00 Very Low Very Low Very Low
Adhesions 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Primers used for GRIA3 SNP were:
Positive_regulation (for) of Gene_expression (used) of GRIA3
1) Confidence 0.49 Published 2010 Journal BMC Med Genet Section Body Doc Link PMC2909201 Disease Relevance 0 Pain Relevance 0
In our present study, microarray analysis and real-time PCR, realized both on BM-MSC and neuron-like cells, have demonstrated an increased expression of ionotropic glutamate receptor AMPA3, also called GRIA3 or GluR3.
Positive_regulation (increased) of Gene_expression (expression) of AMPA3 in neuron associated with glutamate receptor
2) Confidence 0.28 Published 2008 Journal BMC Genomics Section Body Doc Link PMC2358905 Disease Relevance 0 Pain Relevance 0.57

General Comments

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