INT232007

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Context Info
Confidence 0.03
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.65
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

endoplasmic reticulum (Ahsa1) response to stress (Ahsa1) cytoplasm (Ahsa1)
extracellular space (Fasl) signal transduction (Fasl) extracellular region (Fasl)
Ahsa1 (Mus musculus)
Fasl (Mus musculus)
Pain Link Frequency Relevance Heat
addiction 1 50.00 Quite Low
imagery 5 5.00 Very Low Very Low Very Low
agonist 4 5.00 Very Low Very Low Very Low
isoflurane 2 5.00 Very Low Very Low Very Low
Paracetamol 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 3 97.00 Very High Very High Very High
Corneal Neovascularization 2 91.60 High High
Bladder Cancer 91 90.88 High High
Cancer 50 84.72 Quite High
Recurrence 2 84.24 Quite High
Targeted Disruption 15 50.00 Quite Low
Breast Cancer 4 40.32 Quite Low
Reprotox - General 1 8 5.00 Very Low Very Low Very Low
Nicotine Addiction 2 5.00 Very Low Very Low Very Low
Malignant Neoplastic Disease 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In a review, Armstrong et al. [22] summarize a signal-transduction pathway in which a TSP1-stimulated CD36, an 88 kDa transmembrane glycoprotein, phosphorylates kinases p38 and JNK to induce transcription of FasL, which, when bound to Fas, leads to activation of caspases and results in apoptosis.
p38 Positive_regulation (induce) of Transcription (transcription) of FasL associated with apoptosis
1) Confidence 0.03 Published 2008 Journal BMC Urol Section Body Doc Link PMC2374790 Disease Relevance 0.65 Pain Relevance 0

General Comments

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