INT233514

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Context Info
Confidence 0.10
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.45
Pain Relevance 0.05

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Tlr4, Prokr1) plasma membrane (Tlr4, Prokr1) intracellular (Tlr4)
signal transducer activity (Prokr1) cytoplasm (Tlr4)
Tlr4 (Mus musculus)
Prokr1 (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammatory response 12 95.92 Very High Very High Very High
Inflammation 22 48.80 Quite Low
Inflammatory stimuli 8 48.48 Quite Low
chemokine 4 34.96 Quite Low
IPN 2 14.96 Low Low
Analgesic 2 12.80 Low Low
agonist 12 5.00 Very Low Very Low Very Low
adenocard 6 5.00 Very Low Very Low Very Low
antagonist 4 5.00 Very Low Very Low Very Low
cytokine 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 42 95.52 Very High Very High Very High
Mycobacterial Infection 6 84.00 Quite High
Adhesions 2 70.28 Quite High
Cancer 4 66.80 Quite High
Necrosis 4 66.32 Quite High
Repression 2 63.40 Quite High
Stress 2 21.20 Low Low
Injury 2 19.84 Low Low
Inflammatory Pain 2 14.96 Low Low
Hypoxia 2 13.92 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In BMM, Prokr1 was transiently induced by LPS (37-fold at 6 h) but repressed in TEPM (20-fold at 7 h).
LPS Positive_regulation (induced) of Prokr1
1) Confidence 0.10 Published 2008 Journal Immunome Res Section Body Doc Link PMC2394514 Disease Relevance 0.38 Pain Relevance 0.05
The effects of LPS could even be opposing in these two cell populations; Prokr1 was induced 37-fold by LPS in BMM (6 h), but repressed 20-fold in TEPM (7 h) (Figure 3c).
LPS Positive_regulation (induced) of Prokr1
2) Confidence 0.07 Published 2008 Journal Immunome Res Section Body Doc Link PMC2394514 Disease Relevance 0.06 Pain Relevance 0

General Comments

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