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Context Info
Confidence 0.40
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 1.52
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleoplasm (BRCA1, BRCA2) DNA binding (BRCA1, BRCA2) protein complex (BRCA1, BRCA2)
cytoplasm (BRCA1, BRCA2) nucleus (BRCA1, BRCA2) cell cycle (BRCA1, BRCA2)
BRCA1 (Homo sapiens)
BRCA2 (Homo sapiens)
Pain Link Frequency Relevance Heat
cINOD 1 9.16 Low Low
imagery 17 5.00 Very Low Very Low Very Low
addiction 2 5.00 Very Low Very Low Very Low
palliative 2 5.00 Very Low Very Low Very Low
COX-2 inhibitor 1 5.00 Very Low Very Low Very Low
metalloproteinase 1 5.00 Very Low Very Low Very Low
Pain 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Breast Cancer 169 99.96 Very High Very High Very High
Syndrome 13 98.16 Very High Very High Very High
Familial Adenomatous Polyposis 1 92.96 High High
Disease 91 83.84 Quite High
Cancer 111 83.60 Quite High
Microsatellite Instability 1 70.88 Quite High
Fanconi Anemia 2 59.16 Quite High
Colon Cancer 31 40.64 Quite Low
Inflammatory Bowel Disease 21 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
For example, the second susceptibility locus for breast cancer, BRCA2, was mapped by Wooster et al. [37] based on high-risk families for whom BRCA1 linkage was excluded.
BRCA1 Binding (linkage) of BRCA2 associated with breast cancer
1) Confidence 0.40 Published 2008 Journal BMC Med Genet Section Body Doc Link PMC2488325 Disease Relevance 0.77 Pain Relevance 0
We also need to find other proteins that interact with BRCA1 and BRCA2 and elucidate the post-translational modifications that occur as a result of these interactions.
BRCA1 Binding (interact) of BRCA2
2) Confidence 0.32 Published 2008 Journal Breast Cancer Res Section Body Doc Link PMC2397525 Disease Relevance 0.75 Pain Relevance 0

General Comments

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