INT234180

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Context Info
Confidence 0.33
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 1.37
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

DNA binding (BRCA1, BRIP1) cytoplasm (BRCA1, BRIP1) nucleus (BRCA1, BRIP1)
nucleoplasm (BRCA1) chromosome segregation (BRCA1) helicase activity (BRIP1)
BRCA1 (Homo sapiens)
BRIP1 (Homo sapiens)
Pain Link Frequency Relevance Heat
imagery 34 5.00 Very Low Very Low Very Low
palliative 4 5.00 Very Low Very Low Very Low
COX-2 inhibitor 2 5.00 Very Low Very Low Very Low
metalloproteinase 2 5.00 Very Low Very Low Very Low
Pain 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Breast Cancer 334 97.88 Very High Very High Very High
Fanconi Anemia 4 91.32 High High
Cancer 194 80.72 Quite High
Disease 76 73.84 Quite High
Noninfiltrating Intraductal Carcinoma 42 5.00 Very Low Very Low Very Low
Metastasis 30 5.00 Very Low Very Low Very Low
Recurrence 18 5.00 Very Low Very Low Very Low
Adhesions 10 5.00 Very Low Very Low Very Low
Endometriosis (extended) 10 5.00 Very Low Very Low Very Low
Targeted Disruption 8 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
FANCJ encodes BRIP1, which interacts with the BRCT domain of BRCA1 and, of note, mutations in this gene can also cause breast cancer [3].
BRCA1 Binding (interacts) of BRIP1 associated with breast cancer
1) Confidence 0.33 Published 2008 Journal Breast Cancer Res Section Body Doc Link PMC2397525 Disease Relevance 0.68 Pain Relevance 0
FANCJ encodes BRIP1, which interacts with the BRCT domain of BRCA1 and, of note, mutations in this gene can also cause breast cancer [3].
BRCA1 Binding (interacts) of FANCJ associated with breast cancer
2) Confidence 0.28 Published 2008 Journal Breast Cancer Res Section Body Doc Link PMC2397525 Disease Relevance 0.68 Pain Relevance 0

General Comments

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