INT235121

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Context Info
Confidence 0.01
First Reported 2005
Last Reported 2005
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.23

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (GKAP1) Golgi apparatus (GKAP1)
Anatomy Link Frequency
neuronal 1
GKAP1 (Homo sapiens)
Pain Link Frequency Relevance Heat
adenocard 1 96.76 Very High Very High Very High
nMDA receptor 30 93.92 High High
Neurotransmitter 1 93.72 High High
Glutamate 39 92.28 High High
cytokine 5 83.52 Quite High
Inflammation 2 50.00 Quite Low
gABA 28 5.00 Very Low Very Low Very Low
Hippocampus 27 5.00 Very Low Very Low Very Low
5HT 13 5.00 Very Low Very Low Very Low
long-term potentiation 10 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Post-traumatic Stress Disorder 88 50.00 Quite Low
Traumatic Stress Disorders 5 50.00 Quite Low
INFLAMMATION 2 50.00 Quite Low
Neurodegenerative Disease 7 43.28 Quite Low
Drug Induced Neurotoxicity 4 19.12 Low Low
Stress 82 16.48 Low Low
Injury 25 5.00 Very Low Very Low Very Low
Anxiety Disorder 22 5.00 Very Low Very Low Very Low
Cognitive Disorder 9 5.00 Very Low Very Low Very Low
Depression 6 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Important neuronal effects of cGMP include activation of G-kinase, activation or inhibition of phosphodiesterase and subsequent effects on cyclic adenosine monophosphate (cAMP), effects on ion channels and G-proteins, and neurotransmitter release (Harvey 1996; Prast and Philippu 2001).
Positive_regulation (activation) of G-kinase in neuronal associated with neurotransmitter and adenocard
1) Confidence 0.01 Published 2005 Journal Neuropsychiatric Disease and Treatment Section Body Doc Link PMC2413191 Disease Relevance 0 Pain Relevance 0.23

General Comments

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