INT235156

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Context Info
Confidence 0.66
First Reported 2005
Last Reported 2005
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.85
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleoplasm (Atxn1) RNA binding (Atxn1) nucleus (Atxn1)
cytoplasm (Atxn1)
Atxn1 (Mus musculus)
Pain Link Frequency Relevance Heat
Calcium channel 5 5.00 Very Low Very Low Very Low
Migraine 1 5.00 Very Low Very Low Very Low
Kinase C 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Targeted Disruption 8 99.32 Very High Very High Very High
Disease 28 98.54 Very High Very High Very High
Spinocerebellar Ataxia Type 2 17 86.16 High High
Ataxia 33 81.84 Quite High
Retina Disease 3 5.00 Very Low Very Low Very Low
Machado-joseph Disease 3 5.00 Very Low Very Low Very Low
Cerebellar Ataxia 3 5.00 Very Low Very Low Very Low
Death 2 5.00 Very Low Very Low Very Low
Ocular Toxicity (including Many Sub-types) 2 5.00 Very Low Very Low Very Low
Neurodegenerative Disease 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In the case of SCA1, the S776 amino acid is critical in disease development: when changed to A776 the induction of the disease is significantly reduced in transgenic mice, very likely due to lack of phosphorylation at the 776 position in ataxin-1 (Emamian et al 2003).
Phosphorylation (phosphorylation) of ataxin-1 associated with targeted disruption and disease
1) Confidence 0.66 Published 2005 Journal Neuropsychiatric Disease and Treatment Section Body Doc Link PMC2413192 Disease Relevance 0.85 Pain Relevance 0

General Comments

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