INT235181

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Context Info
Confidence 0.00
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.20
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (IRF6, RASGRP1) cytosol (IRF6, RASGRP1) cell differentiation (RASGRP1)
Golgi apparatus (RASGRP1) endoplasmic reticulum (RASGRP1) DNA binding (IRF6)
Anatomy Link Frequency
macrophages 1
IRF6 (Homo sapiens)
RASGRP1 (Homo sapiens)
Pain Link Frequency Relevance Heat
cINOD 2 40.64 Quite Low
corticosteroid 4 33.88 Quite Low
cytokine 52 31.28 Quite Low
Inflammation 10 5.00 Very Low Very Low Very Low
Kinase C 2 5.00 Very Low Very Low Very Low
dexamethasone 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 6 76.40 Quite High
Infection 24 51.20 Quite High
Cancer 6 49.20 Quite Low
INFLAMMATION 12 40.24 Quite Low
Communicable Diseases 2 20.64 Low Low
Aging 2 16.56 Low Low
Necrosis 2 5.00 Very Low Very Low Very Low
Injury 2 5.00 Very Low Very Low Very Low
Stress 2 5.00 Very Low Very Low Very Low
Autoimmune Disease 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Disappearance of the suppressor protein in response to LPS (spot 2Z) suggests that LPS activated the ras pathway.
LPS Positive_regulation (activated) of ras
1) Confidence 0.00 Published 2008 Journal Proteome Sci Section Body Doc Link PMC2413206 Disease Relevance 0.12 Pain Relevance 0
LPS increases active ras-GTP in macrophages [27].
LPS Positive_regulation (increases) of ras in macrophages
2) Confidence 0.00 Published 2008 Journal Proteome Sci Section Body Doc Link PMC2413206 Disease Relevance 0.08 Pain Relevance 0

General Comments

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