INT236392

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Context Info
Confidence 0.00
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.30
Pain Relevance 0.14

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Rad51) nucleus (Rad51) DNA binding (Rad51)
DNA metabolic process (Rad51) cytoplasm (Rad51)
Anatomy Link Frequency
endothelial cells 1
Rad51 (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammatory response 6 97.44 Very High Very High Very High
antagonist 12 65.12 Quite High
Inflammation 9 50.60 Quite High
Inflammatory mediators 1 32.24 Quite Low
cytokine 2 22.08 Low Low
Glutamate 3 5.00 Very Low Very Low Very Low
ketamine 2 5.00 Very Low Very Low Very Low
addiction 2 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Multiple sclerosis 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Congenital Anomalies 3 98.12 Very High Very High Very High
INFLAMMATION 16 97.04 Very High Very High Very High
Gliosis 8 59.16 Quite High
Injury 2 5.00 Very Low Very Low Very Low
Neurological Disease 1 5.00 Very Low Very Low Very Low
Demyelinating Disease 1 5.00 Very Low Very Low Very Low
Toxicity 1 5.00 Very Low Very Low Very Low
Drug Induced Neurotoxicity 1 5.00 Very Low Very Low Very Low
Motor Neuron Diseases 1 5.00 Very Low Very Low Very Low
Disease 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
This autocrine scheme would be consistent with the finding of altered RECA-1 staining patterns (insets, Fig. 3) indicating abnormalities in endothelial cells in QUIN-injected striatum with some recovery provided by Cyclo-VEGI treatment.
Regulation (altered) of RECA-1 in endothelial cells associated with congenital anomalies
1) Confidence 0.00 Published 2008 Journal J Neuroinflammation Section Body Doc Link PMC2430697 Disease Relevance 0.30 Pain Relevance 0.14

General Comments

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