INT23645

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Context Info
Confidence 0.23
First Reported 1986
Last Reported 1989
Negated 0
Speculated 0
Reported most in Abstract
Documents 2
Total Number 2
Disease Relevance 1.18
Pain Relevance 0.44

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Scnn1a) plasma membrane (Scnn1a)
Anatomy Link Frequency
muscle 1
Scnn1a (Mus musculus)
Pain Link Frequency Relevance Heat
antagonist 5 99.50 Very High Very High Very High
Action potential 1 97.74 Very High Very High Very High
carbamazepine 1 81.04 Quite High
headache 1 76.64 Quite High
tetrodotoxin 1 60.12 Quite High
Disease Link Frequency Relevance Heat
Hypopituitarism 8 99.92 Very High Very High Very High
Neuroblastoma 2 99.84 Very High Very High Very High
Congenital Anomalies 1 97.20 Very High Very High Very High
Tinnitus 1 77.44 Quite High
Headache 1 76.64 Quite High
Vomiting 1 75.84 Quite High
Frailty 1 69.68 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Perturbation of glycoprotein processing affects the neurotoxin-responsive Na+ channel in neuroblastoma cells.
Regulation (affects) of Na+ channel associated with neuroblastoma
1) Confidence 0.23 Published 1986 Journal Carbohydr. Res. Section Title Doc Link 2425966 Disease Relevance 0.20 Pain Relevance 0.06
These results indicate that myotonic bursts are related to muscle membrane abnormalities, and each action potential occurs through Na channel, but not through Ca channel Higher dose of calcium antagonist can abolish myotonia by affecting Na channel in addition to their primary effects of Ca channel.
Regulation (affecting) of Na channel in muscle associated with action potential, congenital anomalies, antagonist and hypopituitarism
2) Confidence 0.23 Published 1989 Journal Rinsho Shinkeigaku Section Abstract Doc Link 2791403 Disease Relevance 0.98 Pain Relevance 0.38

General Comments

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