INT236521

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Context Info
Confidence 0.36
First Reported 2008
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 0.07
Pain Relevance 0.09

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Cacna1f)
Cacna1f (Mus musculus)
Pain Link Frequency Relevance Heat
Neurotransmitter 8 64.80 Quite High
Glutamate 5 56.04 Quite High
depression 2 40.76 Quite Low
Calcium channel 6 5.00 Very Low Very Low Very Low
addiction 3 5.00 Very Low Very Low Very Low
Action potential 3 5.00 Very Low Very Low Very Low
long-term potentiation 2 5.00 Very Low Very Low Very Low
anesthesia 2 5.00 Very Low Very Low Very Low
ketamine 1 5.00 Very Low Very Low Very Low
adenocard 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Congenital Anomalies 1 69.08 Quite High
Channelopathies 17 50.00 Quite Low
Depression 2 40.76 Quite Low
Syndrome 6 35.36 Quite Low
Night Blindness 13 29.68 Quite Low
Targeted Disruption 9 27.68 Quite Low
Hypokalemic Periodic Paralysis 29 5.00 Very Low Very Low Very Low
Disease 14 5.00 Very Low Very Low Very Low
Dystonia 10 5.00 Very Low Very Low Very Low
Malignant Hyperthermia 8 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Therefore, we expected that the visual performance of Cacna1fnob2 mice would be greatly reduced or absent, as in Cacna1fG305X mice [37].
Negative_regulation (reduced) of Cacna1fG305X
1) Confidence 0.36 Published 2008 Journal PLoS ONE Section Body Doc Link PMC2432030 Disease Relevance 0 Pain Relevance 0.03
Due to the X-linked condition, CSNB2 results in a complete loss of Cav1.4 channel function only in affected males.
Negative_regulation (loss) of Cav1.4
2) Confidence 0.35 Published 2010 Journal Pflugers Arch Section Body Doc Link PMC2883925 Disease Relevance 0.07 Pain Relevance 0.06

General Comments

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