INT240421

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Context Info
Confidence 0.24
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.34
Pain Relevance 0.37

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

peptidase activity (Ctsa) mitochondrion (Ctsa) nucleus (Ctsa)
lysosome (Ctsa)
Ctsa (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammation 27 97.88 Very High Very High Very High
Inflammatory response 1 76.48 Quite High
cytokine 1 74.04 Quite High
imagery 30 70.88 Quite High
COX2 1 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
isoflurane 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Polyps 40 98.52 Very High Very High Very High
INFLAMMATION 27 97.88 Very High Very High Very High
Apoptosis 1 90.24 High High
Cancer 39 87.48 High High
Inflammatory Bowel Disease 1 86.12 High High
Cleidocranial Dysplasia 15 73.48 Quite High
Targeted Disruption 3 41.20 Quite Low
Familial Adenomatous Polyposis 44 5.00 Very Low Very Low Very Low
Adenoma 9 5.00 Very Low Very Low Very Low
Colon Cancer 7 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
We therefore postulated that if pro-inflammatory cells were the source of cathepsin activity and played a causative role in polyposis, then suppression of polyposis-associated inflammation should hinder progressive polyp growth, and this therapeutic effect should be reflected in a significant down-regulation of cathepsin activity.
Negative_regulation (down) of Negative_regulation (regulation) of cathepsin associated with inflammation and polyps
1) Confidence 0.24 Published 2008 Journal PLoS ONE Section Body Doc Link PMC2488397 Disease Relevance 1.34 Pain Relevance 0.37

General Comments

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