INT241018

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Context Info
Confidence 0.00
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.44
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Sypl) nucleus (Hmbs) cytoplasm (Hmbs)
Anatomy Link Frequency
neuronal 2
Hmbs (Mus musculus)
Sypl (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammation 3 5.00 Very Low Very Low Very Low
tetrodotoxin 1 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Nerve growth factor 1 5.00 Very Low Very Low Very Low
Neurotransmitter 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Diabetes Mellitus 63 92.80 High High
Diabetic Retinopathy 7 5.00 Very Low Very Low Very Low
Body Weight 4 5.00 Very Low Very Low Very Low
Disease 3 5.00 Very Low Very Low Very Low
INFLAMMATION 3 5.00 Very Low Very Low Very Low
Adhesions 2 5.00 Very Low Very Low Very Low
Neurodegenerative Disease 2 5.00 Very Low Very Low Very Low
Hypertension 1 5.00 Very Low Very Low Very Low
Overdose 1 5.00 Very Low Very Low Very Low
Apoptosis 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In agreement with the AT1R-mediated posttranscriptional downregulation of synaptophysin in vivo, in vitro application of angiotensin II to PC12D neuronal cells caused the UPS–mediated degradation of synaptophysin protein via AT1R, which proved to be induced by ERK activation.
UPS Positive_regulation (mediated) of Protein_catabolism (degradation) of synaptophysin protein in neuronal
1) Confidence 0.00 Published 2008 Journal Diabetes Section Abstract Doc Link PMC2494692 Disease Relevance 0.44 Pain Relevance 0

General Comments

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