INT241461

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Context Info
Confidence 0.17
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.49
Pain Relevance 0.06

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular region (TNF, Tnfrsf19) plasma membrane (TNF, Tnfrsf19) signal transduction (TNF)
extracellular space (TNF) intracellular (TNF) extracellular matrix organization (TNF)
TNF (Homo sapiens)
Tnfrsf19 (Mus musculus)
Pain Link Frequency Relevance Heat
Central nervous system 29 68.08 Quite High
dorsal root ganglion 1 51.12 Quite High
Spinal cord 19 41.44 Quite Low
Peripheral nervous system 3 14.92 Low Low
cINOD 6 5.00 Very Low Very Low Very Low
Kinase C 5 5.00 Very Low Very Low Very Low
cva 5 5.00 Very Low Very Low Very Low
ischemia 3 5.00 Very Low Very Low Very Low
adenocard 1 5.00 Very Low Very Low Very Low
Intracerebroventricular 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cancer 2 100.00 Very High Very High Very High
Necrosis 1 100.00 Very High Very High Very High
Shock 2 87.28 High High
Injury 6 67.76 Quite High
Ganglion Cysts 1 51.12 Quite High
Spinal Cord Injury 16 41.44 Quite Low
Targeted Disruption 7 26.16 Quite Low
Disease 18 5.00 Very Low Very Low Very Low
Stroke 15 5.00 Very Low Very Low Very Low
Cv General 3 Under Development 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Further studies revealed that TROY (also known as TAJ), a member of the tumor necrosis factor (TNF) receptor family, formed a functional receptor complex with NgR and Lingo-1, which mediates the inhibitory activity of myelin-associated inhibitors (Park et al 2005; Shao et al 2005).
TNF Binding (complex) of TROY associated with necrosis and cancer
1) Confidence 0.17 Published 2008 Journal Therapeutics and Clinical Risk Management Section Body Doc Link PMC2500253 Disease Relevance 0.49 Pain Relevance 0.06

General Comments

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